❤️ Module 10 · Cardiovascular Part 2

17 drugs · 5 concepts · tested on Exam 6

The classesConceptsDrugsPictures

💡 The big idea

Three cardiac problems in one module: the heart beats WRONG (antiarrhythmics), the heart cannot pump or the coronary supply is short (inotropes, nitrates, antianginals), and the plumbing is furring up (lipid drugs). Rhythm drugs sort into the four Vaughan-Williams classes. Angina drugs all do the same thing from different angles: raise supply or lower demand. Lipid drugs sort by which part of the lipid panel they move.

🧠 How to think about this module

🏷️ The whole module in 11 classes

Learn these groups and the drug list stops being 17 separate names.

ClassWhat it doesExamplesWhat gets tested
Class I — sodium channel blockersSlow conduction through the myocardium.procainamide, quinidine (IA); lidocaine (IB); flecainide (IC)Procainamide causes a lupus-like syndrome and prolongs the QT toward torsades. Continuous ECG during IV loading. Lidocaine toxicity starts with circumoral numbness, metallic taste, and tinnitus, then seizures.
Class II — beta blockersSlow the rate and control the ventricular response in atrial fibrillation.metoprolol, atenolol, esmolol, propranololApical pulse under 60 means hold and call. Bronchospasm with non-selective agents; never stop abruptly.
Class III — potassium channel blockersProlong repolarization; used for serious atrial and ventricular arrhythmias.amiodarone, sotalol, dofetilide, ibutilideAmiodarone damages nearly every organ over time — pulmonary fibrosis, thyroid dysfunction (hypo OR hyper), hepatotoxicity, corneal deposits, blue-gray skin, and photosensitivity. Baseline and periodic chest x-ray, TSH, and liver enzymes.
Class IV — non-dihydropyridine calcium channel blockersSlow the AV node, so they lower both blood pressure and heart rate.verapamil, diltiazemBradycardia, heart block, and hypotension. Verapamil causes marked constipation. Combining with a beta blocker can produce complete heart block — verify before giving.
Dihydropyridine calcium channel blockersPure arterial vasodilators with little effect on heart rate.amlodipine, nifedipine, nicardipine, felodipinePeripheral (ankle) edema, flushing, headache, and reflex tachycardia. Grapefruit juice raises the level. These do NOT control heart rate.
Other antiarrhythmicsDrugs that do not fit the four classes.adenosine, digoxinAdenosine goes as a rapid IV push over 1 to 2 seconds into the port closest to the heart, immediately followed by a fast saline flush; it causes a brief asystolic pause — warn the client and have the crash cart nearby. Digoxin: apical pulse for a full minute, hold if under 60, therapeutic level 0.5 to 2 ng/mL.
ACE inhibitors and ARBs (heart failure and post-MI)Block the RAAS to unload the failing heart and slow remodeling after an MI.captopril, lisinopril, enalapril (ACE); losartan, valsartan (ARB)Dry hacking cough with an ACE inhibitor is the reason to switch to an ARB. Both cause hyperkalemia and angiedema and are contraindicated in pregnancy. Captopril is taken on an empty stomach, 1 hour before meals.
Nitrates and other antianginalsDilate coronary and systemic vessels or reduce myocardial workload.nitroglycerin (SL, patch, IV), isosorbide mononitrate/dinitrate, ranolazineSublingual nitroglycerin: take one tablet, and if the pain is not relieved after that FIRST dose, call 911; the dose may be repeated every 5 minutes up to 3 tablets total. Headache is expected. Remove a nitrate patch for 10 to 12 hours daily to prevent tolerance.
InotropesIncrease the force of contraction in heart failure.digoxin, milrinone, dobutamineDigoxin toxicity risk rises sharply with hypokalemia, so check the potassium. Milrinone is an inodilator — it drops blood pressure, so continuous BP and rhythm monitoring are required.
StatinsBlock cholesterol synthesis in the liver; the biggest LDL reduction available.atorvastatin, simvastatin, rosuvastatin, pravastatinNew muscle pain, tenderness, or weakness with dark cola-colored urine means rhabdomyolysis — stop the drug and check CK. Baseline liver enzymes. Avoid grapefruit with simvastatin and atorvastatin.
Non-statin lipid agentsLower lipids through the gut, the liver, or triglyceride metabolism.cholestyramine (bile acid sequestrant), ezetimibe (absorption inhibitor), gemfibrozil and fenofibrate (fibrates), niacinSequestrants bind other drugs — give everything else 1 hour before or 4 hours after. Fibrates plus a statin raise rhabdomyolysis risk. Niacin causes intense flushing — take aspirin about 30 minutes before and avoid hot drinks and alcohol around the dose.

⚖️ Dihydropyridine vs non-dihydropyridine calcium channel blockers

Dihydropyridines (-dipine)Non-dihydropyridines
Amlodipine, nifedipine, nicardipine, felodipineVerapamil, diltiazem
Act on the blood VESSELS — vasodilationAct on the HEART — slow the AV node
Used for hypertension and vasospastic anginaUsed for rate control in atrial fibrillation and SVT, and for angina
BP goes down; heart rate may reflexively go UPBP goes down AND heart rate goes down
Watch: ankle edema, flushing, headacheWatch: bradycardia, heart block, constipation; dangerous stacked on a beta blocker

🚨 Red flags DANGER

🧵 Exam traps ⭐ HIGH YIELD

🧠 Ways to remember it

🧠 The concepts 5

What is the pharmacological treatment plan for acute coronary syndrome?🚨 DANGER

Immediate treatment of ACS is ASPIRIN (162 to 325 mg CHEWED), NITROGLYCERIN, oxygen if SpO2 is under 90%, and morphine for pain not relieved by nitrates - the classic MONA, given in the order Aspirin, Nitro, Oxygen if needed, Morphine last. Then add a P2Y12 inhibitor, an anticoagulant, a beta blocker, a high-intensity statin, and an ACE inhibitor, and arrange REPERFUSION.

DrugWhyNursing catch
Aspirin 162-325 mg CHEWEDAntiplatelet; biggest mortality benefitChew it. Give it first. Ask about true aspirin allergy
Nitroglycerin SL q5min x3Vasodilation, reduces preload and chest painCheck BP before EACH dose. NEVER with a PDE-5 inhibitor. Hold if SBP under 90 or right ventricular MI
OxygenOnly if SpO2 under 90% or dyspneicRoutine oxygen in a normoxic patient can be harmful
MorphinePain and anxiety refractory to nitratesNow last-line: monitor BP and respirations
P2Y12 inhibitor: ticagrelor, prasugrel, clopidogrelDual antiplatelet therapyBleeding risk; hold before CABG per protocol
Heparin or enoxaparinPrevents clot extensionMonitor aPTT and platelets (HIT)
Beta blocker within 24 hLowers oxygen demand and mortalityHold in acute decompensated HF, bradycardia, hypotension
High-intensity statin, ACE inhibitorPlaque stabilization; remodeling preventionStart regardless of the lipid panel
PCI within 90 min OR fibrinolytic within 30 minREPERFUSION - the definitive treatmentTime is muscle. Screen fibrinolytics for bleeding contraindications

MONA greets everyone, but ASPIRIN goes first and REPERFUSION is what actually saves the muscle.

Which medications cause a reduction in heart rate?⭐ HIGH YIELD

BETA BLOCKERS (metoprolol, atenolol, propranolol, carvedilol), NON-DIHYDROPYRIDINE CALCIUM CHANNEL BLOCKERS (diltiazem, verapamil), DIGOXIN, AMIODARONE, ADENOSINE, and IVABRADINE. Also alpha-2 agonists (clonidine, dexmedetomidine), cholinesterase inhibitors (donepezil), and opioids.

Beta blockers, dilTIAZem and verapamil, digoxin, amiodarone, adenosine. Apical pulse, full minute, hold under 60.

What are the treatment options for atrial fibrillation?⭐ HIGH YIELD

Three goals: RATE control (beta blocker, diltiazem or verapamil, digoxin), RHYTHM control (amiodarone, flecainide, propafenone, sotalol, dofetilide, or electrical cardioversion/ablation), and ANTICOAGULATION to prevent stroke (a DOAC such as apixaban or rivaroxaban, or warfarin), based on the CHA2DS2-VASc score.

GoalOptions
Rate controlBeta blockers (metoprolol, esmolol); diltiazem or verapamil; digoxin (in HF or hypotension)
Rhythm controlAmiodarone, flecainide, propafenone, sotalol, dofetilide; synchronized cardioversion; catheter ablation
Anticoagulation (stroke prevention)DOACs: apixaban, rivaroxaban, dabigatran, edoxaban. Warfarin (INR 2-3) for mechanical valves or mitral stenosis
UNSTABLE patientIMMEDIATE synchronized cardioversion

Rate, Rhythm, Ribbon-thin blood. The anticoagulant is what prevents the stroke.

Review the definitions of inotropy and chronotropy. Identify examples of positive chronotropes, negative chronotropes, positive inotropes, and negative inotropes.⭐ HIGH YIELD

INOTROPY is the FORCE of contraction; CHRONOTROPY is the RATE. POSITIVE INOTROPES: digoxin, dobutamine, dopamine, epinephrine, milrinone. NEGATIVE INOTROPES: beta blockers, diltiazem, verapamil. POSITIVE CHRONOTROPES: atropine, epinephrine, dopamine, isoproterenol. NEGATIVE CHRONOTROPES: beta blockers, diltiazem, verapamil, digoxin, amiodarone, adenosine.

POSITIVE (increases)NEGATIVE (decreases)
INOTROPY (force)digoxin, dobutamine, dopamine, epinephrine, norepinephrine, milrinone, calciumbeta blockers, diltiazem, verapamil, flecainide, high-dose propofol
CHRONOTROPY (rate)atropine, epinephrine, dopamine, isoproterenol, dobutamine, theophyllinebeta blockers, diltiazem, verapamil, DIGOXIN, amiodarone, adenosine, ivabradine, clonidine

Digoxin: squeezes HARDER, beats SLOWER. Everything that slows the rate also weakens the squeeze, except digoxin.

Compare dihydropyridine and non-dihydropyridine calcium channel blockers. Identify examples of medications in each class.⭐ HIGH YIELD

DIHYDROPYRIDINES (the -DIPINES: amlodipine, nifedipine, felodipine, nicardipine, clevidipine) are VASCULAR-selective: they dilate arteries to lower blood pressure and have little effect on the heart. NON-DIHYDROPYRIDINES (verapamil and diltiazem) are CARDIAC-selective: they slow the heart rate, slow AV conduction, and reduce contractility.

Dihydropyridine (-dipine)Non-dihydropyridine
amlodipine, nifedipine, felodipine, nicardipine, clevidipine, nimodipineverapamil, diltiazem
Acts on VESSELS (vascular smooth muscle)Acts on the HEART (SA and AV node, myocardium)
Lowers BP by vasodilation; little effect on rateSlows heart rate, slows AV conduction, reduces contractility
Uses: hypertension, angina, Raynaud's; nimodipine for subarachnoid hemorrhage vasospasmUses: atrial fibrillation and SVT rate control, angina, hypertension
Adverse: ANKLE EDEMA, headache, flushing, REFLEX TACHYCARDIAAdverse: bradycardia, AV block, worsening heart failure; verapamil causes CONSTIPATION
Amlodipine is safe in heart failureAVOID in heart failure with reduced EF and with beta blockers

-DIPINE dilates the PIPEs. Verapamil and diltiazem talk to the HEART.

💉 The drugs 17

💉 AdenosineHIGH ALERT

Antidysrhythmic, Endogenous nucleoside

What it is for

PSVT, as a diagnostic aid to assess myocardial perfusion defects in CAD, Wolff-Parkinson-White syndrome Unlabeled: Wide-complex tachycardia diagnosis

How it works

Slows conduction through AV node, can interrupt reentry pathways through AV node, and can restore normal sinus rhythm in patients with paroxysmal supraventricular tachycardia (PSVT)

Watch for
Teaching

Antidote / reversal: 1

🔗 Full card in the drug guide

💉 AmiodaroneHIGH ALERTBLACK BOX

Antidysrhythmic (class III), Iodinated benzofuran derivative

What it is for

Hemodynamically unstable ventricular tachycardia, supraventricular tachycardia, ventricular fibrillation not controlled by first-line agents

How it works

Prolongs duration of action potential and effective refractory period, noncompetitive α- and β-adrenergic inhibition; increases PR and QT intervals, decreases sinus rate, decreases peripheral vascular resistance

Watch for
Teaching

Antidote / reversal: 1

🔗 Full card in the drug guide

💉 Atenolol/metoprololHIGH ALERTBLACK BOX

Antihypertensive, antianginal, β-Blocker, β1-, β2-blocker (high doses)

What it is for

Hypertension, angina pectoris; suspected or known MI (IV use); MI prophylaxis

How it works

Competitively blocks stimulation of β-adrenergic receptor within vascular smooth muscle; produces negative chronotropic activity (decreases rate of SA node discharge, increases recovery time), slows conduction of AV node …

Watch for

Antidote / reversal: 1

🔗 Full card in the drug guide

💉 CaptoprilBLACK BOX

Antihypertensive, Angiotensin-converting enzyme (ACE) inhibitor

What it is for

Hypertension, HF, left ventricular dysfunction after MI, diabetic nephropathy, proteinuria, acute MI, hypertensive emergency/urgency,

How it works

Selectively suppresses reninangiotensin-aldosterone system; inhibits ACE; prevents conversion of angiotensin I to angiotensin II

Watch for
Teaching
🔗 Full card in the drug guide

💉 Digoxin (What are the signs/symptoms and risk factors for toxicity?)HIGH ALERT

Cardiac glycoside, inotropic, antidysrhythmic, Digoxin preparation

What it is for

Heart failure, atrial fibrillation/flutter, paroxysmal supraventricular tachycardia (PSVT) treatment/prophylaxis

How it works

Inhibits the sodium-potassium ATPase pump, which makes more calcium available for contractile proteins, thereby resulting in increased cardiac output (positive inotropic effect); increases force of contractions …

Watch for
Teaching

Antidote / reversal: 1

🔗 Full card in the drug guide

💉 LosartanBLACK BOX

Antihypertensive, Angiotensin II receptor (type AT1) antagonist

What it is for

Hypertension, alone or in combination; nephropathy in type 2 diabetes; proteinuria; stroke prophylaxis for hypertensive patients with left ventricular hypertrophy

How it works

Blocks the vasoconstrictor and aldosterone-secreting effects of angiotensin II; selectively blocks the binding of angiotensin II to the AT1 receptor found in tissues

Watch for
Teaching
🔗 Full card in the drug guide

💉 MilrinoneHIGH ALERT

Inotropic/vasodilator agent, Bipyridine phosphodiesterase inhibitor

What it is for

Short-term management of advanced heart failure that has not responded to other medication

How it works

Positive inotropic agent; increases contractility of cardiac muscle with vasodilator properties; reduces preload and afterload by direct relaxation on vascular smooth muscle

Watch for
Teaching

Antidote / reversal: 1

🔗 Full card in the drug guide

💉 Nifedipine/amlodipine

Calcium channel blocker, antianginal, antihypertensive, Dihydropyridine

What it is for

Chronic stable angina pectoris, variant angina, hypertension

How it works

Inhibits calcium ion influx across cell membrane during cardiac depolarization; relaxes coronary vascular smooth muscle; dilates coronary arteries; increases myocardial oxygen delivery in patients with vasospastic angina …

Watch for
Teaching

Antidote / reversal: 1

🔗 Full card in the drug guide

💉 Nitroglycerin

Antianginal, Nitrate, vasodilator

What it is for

Acute angina and prevention of angina; acute coronary syndrome and MI; heart failure with pulmonary congestion; hypertension during surgery.

How it works

Turns into nitric oxide in the vessel wall, which relaxes smooth muscle. Veins dilate first, so less blood comes back to the heart — preload drops and the heart has less work to do. At higher levels arteries dilate too, and it opens the coronary arteries directly.

Watch for
Teaching
🔗 Full card in the drug guide

💉 ProcainamideHIGH ALERTBLACK BOX

Antidysrhythmic (class IA), Procaine HCl amide analog

What it is for

Life-threatening ventricular dysrhythmias

How it works

Depresses excitability of cardiac muscle to electrical stimulation and slows conduction velocity in atrium, bundle of His, and ventricle; increases refractory period

Watch for
Teaching

Antidote / reversal: 1

🔗 Full card in the drug guide

💉 Ranolazine

Antianginal, Piperazine derivative

What it is for

Chronic angina pectoris

How it works

Antianginal, antiischemic; unknown, may work by inhibiting portal fatty-acid oxidation

Watch for
Teaching
🔗 Full card in the drug guide

💉 Verapamil/diltiazemHIGH ALERT

Calcium channel blocker; antihypertensive; antianginal, antidysrhythmic (class IV), Diphen

What it is for

Chronic stable, vasospastic, unstable angina; dysrhythmias, hypertension, supraventricular tachycardia, atrial flutter or fibrillation Unlabeled uses: Prevention of migraines

How it works

Inhibits calcium ion influx across cell membrane during cardiac depolarization; produces relaxation of coronary vascular smooth muscle; dilates coronary arteries; decreases SA/AV node conduction; dilates peripheral arteries

Watch for
Teaching

Antidote / reversal: 1

🔗 Full card in the drug guide

💉 Atorvastatin

Antilipidemic, HMG-CoA reductase inhibitor (statin)

What it is for

As adjunct for primary hypercholesterolemia (types Ia, Ib), elevated triglyceride levels, prevention of CV disease by reduction of heart risk in those with mildly elevated cholesterol, heterozygous familial hypercholesterolemia in pediatric patients …

How it works

Inhibits HMG-CoA reductase enzyme, which reduces cholesterol synthesis; high doses lead to plaque regression

Watch for
Teaching
🔗 Full card in the drug guide

💉 Cholestyramine

Antilipemic, Bile acid sequestrant

What it is for

Primary hypercholesterolemia (esp. type IIa/IIb hyperlipoproteinemia), pruritus associated with biliary obstruction

🔗 Full card in the drug guide

💉 Ezetimibe

Antilipemic; cholesterol absorption inhibitor

What it is for

Hypercholesterolemia, homozygous familial hypercholesterolemia (HoFH), homozygous sitosterolemia

How it works

Inhibits absorption of cholesterol by the small intestine, causes reduced hepatic cholesterol stores

Watch for
Teaching
🔗 Full card in the drug guide

💉 Gemfibrozil

Antilipemic, Fibric acid derivative

What it is for

For use as an adjunct to diet for the treatment of hyperlipoproteinemia and for hypertriglyceridemia including type IV (elevated triglycerides, VLDL) and type V (elevated triglycerides, chylomicrons …

How it works

Inhibits biosynthesis of VLDL, decreases triglycerides, production in the liver increases HDL

Watch for
Teaching
🔗 Full card in the drug guide

💉 Niacin (vitamin B3/nicotinic acid)

Vit B3, antihyperlipidemic, Water-soluble vitamin

What it is for

Pellagra, hyperlipidemias (types 4, 5), peripheral vascular disease that presents a risk for pancreatitis

🔗 Full card in the drug guide
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Where this came from. The drug cards come from your own drug guide, fact-checked against FDA labeling. The explanations were written from your course textbook, Pharmacology (WTCS, 2e). If anything here contradicts your instructor, believe your instructor — they write the exam.
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