Most childhood rashes are identified by what came before the rash β the prodrome,
where it starts, and how it spreads.
Illness
Signature
Precautions
Measles (rubeola)
Koplik spots in the mouth, then rash from the face down; 3 Cs β cough, coryza, conjunctivitis
Airborne
Varicella
Lesions in all stages at once, intensely itchy
Airborne + contact
Pertussis
Paroxysmal cough with an inspiratory whoop
Droplet
Fifth disease
βSlapped cheekβ, then lacy rash on limbs
Standard once rash appears
Scarlet fever
Sandpaper rash, strawberry tongue, strep throat
Droplet; needs antibiotics
🖼️ Pediatric medication administration.Swipe it sideways if it is cut off, or tap to open it full size.
π¨ Two absolutes
Never give aspirin to a child with a viral illness - Reye syndrome. Acetaminophen or ibuprofen instead.
Untreated strep throat can become rheumatic fever and damage heart valves. Finish the whole antibiotic course.
β Pediatric burns β what differs from adults
Children have proportionally larger heads, so adult Rule of Nines under-estimates
β use a pediatric chart
Thinner skin β deeper burns from lower temperatures
Larger surface area to body mass β faster fluid loss and heat loss
Burn patterns suggest abuse: stocking or glove distribution, sharp
waterline edges, cigarette circles, or a story that does not match the injury
🖼️ Acute respiratory failure, type I versus type II.Swipe it sideways if it is cut off, or tap to open it full size.
π‘οΈ Suspected abuse β what the nurse does
Document objectively in the childβs own words and with exact measurements.
Report β nurses are mandatory reporters, and suspicion is enough; proof is not required.
Do not confront the caregiver or promise the child secrecy. Keep the child safe and report.
β High-yield β what the exam actually asks
Show 5 moreHide these 5
HIV: CD4 tracks disease, <200 defines category C/AIDS. PCR/viral culture diagnoses infants; ELISA plus Western blot after 18 months. Standard precautions only. Live vaccines need caution.
Contagion endpoints: varicella until every lesion crusts; rubeola 4 days after rash onset; fifth disease is no longer contagious once the rash appears.
Rubeola: cough, coryza, conjunctivitis, plus Koplik spots.
Show 5 moreHide these 5
Mono: splenomegaly means no contact sports for about 4 weeks. Monospot positive. Recovery takes months.
Impetigo: honey-colored crusts, mupirocin. Head lice: 1% permethrin plus nit combing, repeat in 2 weeks. Pinworms: tape test, mebendazole for the whole household.
Burns: airway first, then fluids, then wound care and pain. Tepid soaks, never ice. Do not drain blisters. Children's thinner skin means deeper injury for the same exposure.
Accutane is teratogenic β contraception is mandatory. Antibiotics can reduce oral contraceptive efficacy.
Diaper dermatitis: zinc barrier and air exposure. Never talc.
Rotavirus has hard age limits. The series must start before 15 weeks 0 days and the last dose is given by 8 months 0 days. Miss the window and it is not given at all.
Hepatitis B at birth, but it is withheld from a preterm infant under 2,000 g if the mother is hepatitis B negative.
Burn depth by healing time: superficial partial thickness heals in 5–10 days, deep partial thickness in under 21 days, and full thickness takes longer than 21 days.
Burn care teaching: mild soap and tepid water without scrubbing. No greasy lotions, no butter. Resuscitation fluid is 0.9% sodium chloride.
The burn phases in order: emergent/resuscitative (shock and airway), acute, then rehabilitative, which begins once the wound is closed and is about preventing contractures and scarring.
📚 From your Maternal & Child textbook
Pillitteri, Maternal and Child Health Nursing — ch. 42 (immune) · ch. 43 (infectious) · ch. 52 (unintentional injury).
Type I hypersensitivity is immediate and IgE-mediated — anaphylaxis, allergic asthma. Type IV is delayed and cell-mediated, which is why a TB skin test is read at 48 to 72 hours.
β οΈ Exam traps
Airborne vs droplet assignment is the single most tested item here. Learn the grid cold.
Varicella itches and starts on the trunk; rubeola and rubella do not itch.
Neutropenic clients need protective isolation β the opposite direction from airborne isolation, even though both use a private room.
π§ Mind maps 6
One per disorder, built from the structure of your ATI chapter.
Skin Infections and Infestations
π― Who gets it
Bacterial: contact with an infected person, congenital or acquired immunodeficiency, immunosuppression
Fungal: geographic area
Arthropod bites: dark clothing and exposed skin
Scabies: continuous close personal contact; anyone can develop scabies or lice
π What you see
Impetigo contagiosa (Staphylococcus, Streptococcus): a reddish macule turns vesicular and ruptures readily, leaving moist erosion whose secretions dry into honey-colored crusts; it commonly itches and extends outward and by direct contact
Verruca (warts, human papillomavirus): elevated, rough, gray-brown firm papules anywhere on the skin, single or grouped
Verruca plantaris: flat warts on the plantar surface, sometimes surrounded by hyperkeratosis
Herpes simplex type 1 and 2: cold sore or fever blister near mucocutaneous areas β lips, nose, buttocks, genitalia
π§ͺ What confirms it
Bacterial, viral, and fungal cultures; scabies confirmed by microscopic examination
Atopic dermatitis: existing allergic condition and family history of atopy, previous skin disorder or current flare, exposure to irritants, genetic predisposition, geographic location
Acne: genetic link, more common in males, typical during adolescence, hormonal fluctuations trigger flares
π What you see
Contact dermatitis: red bumps that can become moist weeping blisters, skin warm and tender, oozing, drainage, or crusts, skin becoming scaly, raw, or thickened
Diaper dermatitis: change diapers frequently and remove soiled diapers promptly
Cleanse the diaper area with a nonirritating cleanser or plain warm water using soft cloths or alcohol- and fragrance-free wipes
Expose the affected area to air and apply a skin barrier such as zinc oxide, which should not be washed off at each change
Contact dermatitis: remove the irritant and limit further exposure
π Drugs
Hydroxyzine or diphenhydramine for allergic or medication reactions and severe pruritus β teach scheduled administration and supervise the child because of sedation
Loratadine or fexofenadine as a nonsedating oral antihistamine preferred for daytime use
Antibiotics for secondary infection β finish the full course
Clotrimazole and other antifungal ointments for Candida albicans in diaper dermatitis
π¬ What you teach
Avoid bubble baths and harsh soaps
Wear long sleeves and pants when exposure to irritants is likely and remove an offending agent immediately after contact
Wash the face gently without scrubbing or abrasive cleansers
β οΈ What goes wrong
Bacterial infection through breaks in the skin from scratching β monitor for infection, keep nails short, cleanse with mild soap and water, give antipruritics and antibiotics, and teach avoidance of offending agents
Psychosocial impairment from acne with stress, lowered self-esteem, and depression over appearance β monitor mood and behavior, encourage the adolescent to talk about feelings, support adherence, and have caregivers report mood changes
Skin scarring from scratching or delayed acne treatment, either atrophic with tissue loss or hypertrophic such as keloids β managed with topical medications, injectable steroids, silicone dressings, punch excision, cryosurgery, dermabrasion, or microdermabrasion, with realistic reassurance that therapy improves appearance
Read left to right: who gets it β what you see β what confirms it β what you do β what goes wrong. Cover a column and rebuild it out loud.
Communicable Diseases
π― Who gets it
Prior communicable disease history and recent exposure to a known case
Not immunized or behind on immunizations
Immunocompromised status and chronic illness
Crowded living conditions, poor sanitation, poor nutrition
π What you see
Conjunctivitis: pink or red sclera, swollen conjunctiva, excessive tearing; bacterial adds yellow-green purulent discharge and morning eyelid crusting, viral produces watery discharge
Fifth disease prodrome: several days of fever, runny nose, headache
Fifth disease rash lasting 7 days to several weeks: red slapped-cheek facial rash on days 1-4, then symmetric maculopapular red spots spreading proximal to distal on the extremities over a week, with a secondary itchy rash possible elsewhere including the soles
Measles prodrome 3-4 days before the rash: mild to moderate fever, cough, runny nose, red eyes, sore throat
π§ͺ What confirms it
CBC, electrolyte panel, and mono spot test for infectious mononucleosis
Pertussis in infants and children: seizures, apnea, pneumonia, ear infection, encephalopathy, hemorrhage, weight loss, hernia, and death; in teens and adults: rib fracture, syncope, pneumonia, weight loss, and loss of bladder control
Read left to right: who gets it β what you see β what confirms it β what you do β what goes wrong. Cover a column and rebuild it out loud.
Acute Otitis Media
π― Who gets it
Children's eustachian tubes are shorter and more horizontal than adults'; incidence peaks in the first 24 months and declines after age 5
Provide comfort measures and diversional activities
Position the child upright
Manage fever
π Drugs
Acetaminophen or ibuprofen for analgesia and fever
Antibiotics: amoxicillin, amoxicillin-clavulanate, or azithromycin orally for 10 days, or IM ceftriaxone for three doses
Antibiotics are recommended over 6 months of age for severe symptoms β increased pain or temperature above 39 C (102.2 F) β lasting more than 2 days, and for children 6-23 months with bilateral disease
Benzocaine ear drops relieve pain
π¬ What you teach
Finish the entire antibiotic course and watch for allergy such as rash or difficulty breathing
Feed the child upright when bottle- or breastfeeding
Apply antibiotic ear drops as prescribed and use comfort measures
β οΈ What goes wrong
Hearing loss and speech delay β assess and monitor for deficits, refer for audiology testing, and arrange speech therapy if needed
Read left to right: who gets it β what you see β what confirms it β what you do β what goes wrong. Cover a column and rebuild it out loud.
HIV/AIDS
π― Who gets it
Perinatal transmission or ingestion of breast milk from a person with HIV
Exposure to blood products or body fluids containing the virus
Sexual assault
Risky behaviors such as unprotected sex and IV substance use
π What you see
Immunologic staging by CD4 count under 12 months: stage 1 is 1,500 cells/microliter or more, stage 2 is 750-1,499, stage 3 is under 750
Ages 1-5 years: stage 1 is 1,000 or more, stage 2 is 500-999, stage 3 is under 500
Age 6 and older: stage 1 is 500 or more, stage 2 is 200-499, stage 3 is under 200
Clinical category N is not symptomatic β no HIV-attributable findings or only one mildly symptomatic condition
π§ͺ What confirms it
18 months and older: positive ELISA confirmed by Western blot
Under 18 months born to an infected parent: positive polymerase chain reaction test
Rapid saliva testing detects antibodies noninvasively but a positive result requires blood confirmation
Non-nucleoside reverse transcriptase inhibitors β delavirdine, efavirenz, nevirapine, doravirine, etravirine, rilpivirine β bind viral DNA for direct inhibition
π¬ What you teach
Take medications on a regular schedule without missed doses; the illness is chronic and treatment is lifelong
Watch for adverse effects and use strategies to lessen them
Call the provider for headache, fever, lethargy, warmth, tenderness or redness at joints, or neck stiffness
β οΈ What goes wrong
Failure to thrive β obtain baseline height and weight and monitor, promote optimal nutrition including total parenteral nutrition if needed, assess growth and development for delays, and provide age-appropriate toys and peer play
Pneumocystis pneumonia β monitor respiratory rate and effort, oxygen saturation, and breath sounds, give antibiotics, antipyretics, and analgesics, maintain hydration and fluid and electrolyte balance, promote rest, and teach infection prevention and medication adherence
Read left to right: who gets it β what you see β what confirms it β what you do β what goes wrong. Cover a column and rebuild it out loud.
Burns
π― Who gets it
Abuse or neglect
Inadequate supervision
π What you see
Determine which agent burned the child (electrical, chemical, moist heat, dry heat, ionizing radiation), how long contact lasted, and which body area was involved
Younger children sustain deeper injuries because their skin is thinner
Extent measured as total body surface area using age-related charts and expressed as a percentage
Severity depends on age, causative agent, body area, and burn extent and depth
π§ͺ What confirms it
Major burns: CBC, electrolytes, BUN, ABGs, random glucose, liver enzymes, urinalysis
On-site: stop the burning process and remove the child from the source
Lay the child flat and smother flames by rolling them in a rug or blanket, or roll the child slowly if nothing is available
Remove clothing and jewelry that conduct heat
Apply tepid water soaks or run water over the injury β never ice, which risks hypothermia
π Drugs
Topical agents for partial-thickness burns β silver sulfadiazine (inhibits bacterial growth), mafenide acetate, collagenase (santyl), bacitracin β applied to a cleansed, debrided area with sterile gloves
Topical prepackaged mesh gauze dressings protect the wound, improve comfort, speed healing, and reduce dressing change frequency
Morphine sulfate IV for analgesia, sometimes premedicated before dressing changes β monitor for respiratory depression and discuss dependency risk with prolonged use
Midazolam, fentanyl, propofol, and nitrous oxide for sedation and analgesia given IV just before a procedure
π¬ What you teach
Continue range-of-motion exercises and physical therapy to prevent contractures
Assess the wound for infection and perform wound care at home
Home safety: cover electrical outlets, supervise bathing, keep irons out of reach, and teach the danger of playing with matches
β οΈ What goes wrong
Direct thermal injury with face and lip burns damages the tracheobronchial tree after inhaling heated gases and toxic combustion products, and can be delayed 24-72 hr β wheezing, increased secretions, hoarseness, wet rales, singed nasal hairs, laryngeal edema, carbonaceous secretions
Carbon monoxide injury occurs in enclosed spaces, causing mental status change and dyspnea β maintain airway and ventilation with 100% oxygen
Pulmonary injury from inhaled fumes, vapors, gases, and smoke β maintain the airway with intubation or sometimes tracheostomy and give oxygen
Hypertrophic scarring from collagen deposition during healing appears raised β apply uniform pressure, fit custom pressure garments, continue physical therapy, give cetirizine or diphenhydramine for itching, and massage and moisturize scars as prescribed
Read left to right: who gets it β what you see β what confirms it β what you do β what goes wrong. Cover a column and rebuild it out loud.
🎥 Lecture recordings 2
Tap a card to open that recording in Google Drive. The same list lives in the lecture library.
ATI Active Learning Template β System DisorderSkin Infections and Infestations
Filled from ATI chapter 30, row by row from that chapterβs own sections β 12 of 12 rows have content.
π§ What it isAlterations in Health (Diagnosis) Β· Health Promotion & Disease Prevention
Alterations in Health (Diagnosis)
Skin infections may be bacterial (impetigo contagiosa, pyoderma, folliculitis, furuncle, carbuncle, cellulitis, staphylococcal scalded skin syndrome), viral (verruca, verruca plantaris, herpes simplex, varicella zoster, molluscum contagiosum), or fungal (tinea capitis, corporis, cruris, pedis/unguium, candidiasis). Bites and stings come from bees, fire ants, mosquitoes, flies, chiggers, ticks, mites, spiders, and scorpions. Infestations include scabies and lice.
Health Promotion & Disease Prevention
From this module β built from the notes above on this page, not a section of the ATI chapter.
Handwashing is the single most effective measure β these spread by contact.
No sharing of hats, combs, bedding, towels or sports equipment.
Keep nails short; treat itch, because scratching is what turns an infestation into a bacterial infection.
Prompt treatment of household contacts, or it cycles round the family indefinitely.
Return-to-school rules differ by condition β check rather than assume.
π How it shows upAssessment β Risk Factors Β· Assessment β Expected Findings
Assessment β Risk Factors
Bacterial: contact with an infected person, congenital or acquired immunodeficiency, immunosuppression
Fungal: geographic area
Arthropod bites: dark clothing and exposed skin
Scabies: continuous close personal contact; anyone can develop scabies or lice
Assessment β Expected Findings
Impetigo contagiosa (Staphylococcus, Streptococcus): a reddish macule turns vesicular and ruptures readily, leaving moist erosion whose secretions dry into honey-colored crusts; it commonly itches and extends outward and by direct contact
Verruca (warts, human papillomavirus): elevated, rough, gray-brown firm papules anywhere on the skin, single or grouped
Verruca plantaris: flat warts on the plantar surface, sometimes surrounded by hyperkeratosis
Herpes simplex type 1 and 2: cold sore or fever blister near mucocutaneous areas β lips, nose, buttocks, genitalia
Scabies (Sarcoptes scabiei): intense itching especially at night, rash between the fingers and in popliteal folds and inguinal regions, thin pencil-like burrow marks with the mite visible as a black dot at the end of a grayish-brown burrow; infants show widespread involvement with pimples on the trunk
Head lice (Pediculus humanus capitis): adult lice are small, grayish-tan, wingless, and hard to see; nits resemble dandruff but are firmly attached to the hair shaft
Lyme disease stage 1, 3-30 days after the bite: red macular/papular lesion expanding into a red bull's-eye rash, with chills, fever, itching, headache, stiff neck, muscle weakness
Lyme stage 2, 3-12 weeks after the bite: neurologic, cardiac, and musculoskeletal involvement with facial paralysis or weakness, muscle pain, fever, fatigue
π§ͺ How it is confirmedLaboratory Tests Β· Diagnostic Procedures
Laboratory Tests
Bacterial, viral, and fungal cultures; scabies confirmed by microscopic examination
Diagnostic Procedures
From this module β built from the notes above on this page, not a section of the ATI chapter.
Most are diagnosed on appearance and distribution.
Wood lamp β some tinea capitis fluoresces.
KOH preparation of skin scrapings for fungal hyphae.
Bacterial culture of pus for impetigo not responding to treatment, or for suspected MRSA.
Visual inspection for live lice and nits within 6 mm of the scalp β nits further out are old and not active infestation.
Verruca plantaris: apply caustic solution, wear insoles with holes to relieve pressure for 2-3 days, soak the area 20 min, and repeat until the wart falls off
Tick bites: observe the child for 30 days; a single antibiotic dose for those meeting criteria and a 2-3 week course for confirmed disease
Lyme antibiotics: doxycycline over age 8, amoxicillin or cefuroxime under age 8, and cefuroxime for penicillin allergy
Provide cool baths or compresses and keep fingernails clean and trimmed
Medications
From this module β built from the notes above on this page, not a section of the ATI chapter.
Impetigo: topical mupirocin for limited disease; oral antibiotics if widespread.
Tinea corporis: topical antifungal. Tinea capitis needs oral antifungal β topical will not reach the follicle.
Head lice: permethrin or a dimeticone, repeated at the interval on the label to catch newly hatched lice.
Scabies: permethrin 5% from the neck down, left on 8β14 hours; treat all household contacts on the same day.
Itching after successful scabies treatment can persist for weeks and does not mean it failed β retreating unnecessarily causes dermatitis.
Therapeutic Procedures
From this module β built from the notes above on this page, not a section of the ATI chapter.
Wet-to-dry or soak-and-smear to lift crusts in impetigo before topical treatment.
Wash bedding and clothing hot and dry hot; bag non-washables for 72 hours.
Wet combing with a fine-toothed nit comb, repeated every few days.
Vacuum furniture and car seats; treat pets for ringworm where they are the source.
Cool compresses and emollients for itch.
π¬ Around the patientClient Education Β· Interprofessional Care
Client Education
Use good hand hygiene and take steps to prevent spread
Do not share clothing, hats, combs, brushes, or towels, especially with fungal infections
Do not squeeze vesicles
Apply topical medications as prescribed and watch for adverse effects
Avoid home remedies, which can worsen the infection
Launder potentially contaminated clothing and bedding correctly, and seal items that cannot be washed in a tight bag for 14 days
Disinfect combs, brushes, and hair accessories by boiling 10 min or soaking 1 hr in a lice-killing product
Interprofessional Care
From this module β built from the notes above on this page, not a section of the ATI chapter.
Provider for prescriptions and for a diagnosis that is not resolving.
School nurse for exclusion policy and for identifying an outbreak.
Public health for outbreaks in nurseries and schools.
Social work where laundry facilities, bedding or the cost of treatment are the real barrier.
β οΈ What goes wrongComplications
Complications
From this module β built from the notes above on this page, not a section of the ATI chapter.
Secondary bacterial infection from scratching β cellulitis or impetigo on top of the original problem.
Post-streptococcal glomerulonephritis after streptococcal impetigo β watch for dark urine and puffiness.
Permanent scarring or hair loss with severe tinea capitis or a kerion.
School exclusion, missed days and social stigma β a real complication for the child.
Reinfestation when contacts are not treated together.
ATI Active Learning Template β System DisorderDermatitis and Acne
Filled from ATI chapter 31, row by row from that chapterβs own sections β 12 of 12 rows have content.
π§ What it isAlterations in Health (Diagnosis) Β· Health Promotion & Disease Prevention
Alterations in Health (Diagnosis)
Contact dermatitis is an inflammatory hypersensitivity reaction when skin meets an irritant such as feces, urine, soaps, poison ivy, animals, metals, dyes, or medications. Diaper dermatitis follows prolonged contact with urine at increased pH, stool, friction, chemicals, soaps, or detergents. Seborrheic dermatitis (cradle cap, blepharitis, otitis externa) has an unknown cause and peaks in infancy and puberty. Atopic dermatitis is a form of eczema marked by pruritus and associated with allergy history; it is classified by age, lesion distribution, and appearance, and cannot be cured but can be well controlled. Acne vulgaris is the most common adolescent skin condition, involving pilosebaceous follicles of the face, neck, chest, and upper back; it is self-limiting and not dangerous but threatens self-image, with Cutibacterium acnes driving the inflammation.
Health Promotion & Disease Prevention
From this module β built from the notes above on this page, not a section of the ATI chapter.
Eczema: moisturise generously and daily, even when the skin is clear β that is the treatment, not the aftercare.
Short lukewarm baths, then moisturise within 3 minutes to trap water in the skin.
Avoid known triggers: soap, fragrance, wool, overheating, and identified food triggers.
Acne: gentle cleansing twice daily. Scrubbing and picking make it worse and cause scarring.
Correct the myths out loud β acne is not caused by dirt or chocolate.
π How it shows upAssessment β Risk Factors Β· Assessment β Expected Findings
Atopic dermatitis: existing allergic condition and family history of atopy, previous skin disorder or current flare, exposure to irritants, genetic predisposition, geographic location
Acne: genetic link, more common in males, typical during adolescence, hormonal fluctuations trigger flares
Acne: cosmetics containing petrolatum and lanolin, cooking grease exposure in fast-food work, and a possible dietary link to high glycemic index foods and dairy
Assessment β Expected Findings
Contact dermatitis: red bumps that can become moist weeping blisters, skin warm and tender, oozing, drainage, or crusts, skin becoming scaly, raw, or thickened
Atopic dermatitis: small diffuse areas of hypopigmentation, pallor around nose, mouth, and ears, bluish discoloration under the eyes, lymphadenopathy near affected areas
Atopic dermatitis distribution: generalized on cheeks and scalp in infants; flexural areas (antecubital and popliteal fossae, neck) and wrists in children
Acne: open comedones (blackheads) and closed comedones (whiteheads) on face, neck, back, and chest, with inflammation producing papules, pustules, nodules, or cysts, and a pattern of exacerbation and remission
π§ͺ How it is confirmedLaboratory Tests Β· Diagnostic Procedures
Laboratory Tests
From this module β built from the notes above on this page, not a section of the ATI chapter.
Usually none β both are clinical diagnoses.
Allergy testing only where a specific trigger is genuinely suspected in eczema.
Bacterial culture where eczema is infected or acne is not responding.
Isotretinoin requires monitoring: pregnancy tests, lipids and liver enzymes, on a strict schedule.
Diagnostic Procedures
From this module β built from the notes above on this page, not a section of the ATI chapter.
Diagnosed by appearance and distribution β eczema follows the flexures in older children, the face and extensors in infants.
Patch testing for suspected contact dermatitis.
Assess severity by extent, sleep disturbance and effect on daily life, not by appearance alone.
Photograph with consent to track response over time.
Diaper dermatitis: change diapers frequently and remove soiled diapers promptly
Cleanse the diaper area with a nonirritating cleanser or plain warm water using soft cloths or alcohol- and fragrance-free wipes
Expose the affected area to air and apply a skin barrier such as zinc oxide, which should not be washed off at each change
Contact dermatitis: remove the irritant and limit further exposure
Poisonous plant exposure: flush the area with cold running water as soon as possible and wash clothes and shoes in hot water with detergent
Apply calamine lotion, Burow solution compresses, or colloidal oatmeal baths, plus topical corticosteroid gel; oral corticosteroids for severe reactions or involvement of face, neck, or genitalia
Seborrheic dermatitis: daily scalp and hair hygiene, gently scrub to lift scaly crusted patches, soften overnight with petrolatum or mineral oil, remove loosened crusts with a fine-tooth comb, and use a daily antiseborrheic shampoo containing sulfur and salicylic acid
Atopic dermatitis: apply overnight wet wraps for severe pruritus by covering the area with water-moistened gauze then dry gauze on top
Acne: discuss the disease process with the adolescent and family and stress adherence to the plan of care
Medications
Hydroxyzine or diphenhydramine for allergic or medication reactions and severe pruritus β teach scheduled administration and supervise the child because of sedation
Loratadine or fexofenadine as a nonsedating oral antihistamine preferred for daytime use
Antibiotics for secondary infection β finish the full course
Clotrimazole and other antifungal ointments for Candida albicans in diaper dermatitis
Topical corticosteroids intermittently to control flares, chosen as low, moderate, or high potency based on the site, extent of involvement, and child's age; low potency is safe on the face
Tacrolimus or pimecrolimus for children over 2 years, started at the beginning of an atopic dermatitis flare when the skin reddens and itches
Benzoyl peroxide inhibits C. acnes and can bleach linens, towels, and clothing but not skin
Topical and oral antibacterials such as clindamycin, azelaic acid, and dapsone inhibit C. acnes β monitor for allergic reaction
Therapeutic Procedures
From this module β built from the notes above on this page, not a section of the ATI chapter.
Emollients, wet wrap therapy for severe flares, and bleach baths where recommended.
Topical corticosteroid to the affected area only for as long as needed β the lowest strength that works.
Cotton clothing, nails short, cotton mittens at night for infants.
Acne: topical retinoid, benzoyl peroxide, or antibiotics; isotretinoin for severe nodulocystic disease.
Address the psychological effect of acne directly β it correlates with depression more strongly than its severity suggests.
π¬ Around the patientClient Education Β· Interprofessional Care
Client Education
Avoid bubble baths and harsh soaps
Wear long sleeves and pants when exposure to irritants is likely and remove an offending agent immediately after contact
Wash the face gently without scrubbing or abrasive cleansers
Keep nails short and trimmed, and put gloves or cotton socks on the hands for sleeping
Clothe young children in one-piece cotton outfits that are soft and cover the arms and legs
Wash clothes and linens in mild detergent and repeat the wash cycle without soap
Change diapers as soon as they are wet or soiled and watch for signs of infection
Interprofessional Care
From this module β built from the notes above on this page, not a section of the ATI chapter.
Dermatologist for severe or refractory disease and for isotretinoin.
Allergist where food or environmental triggers are suspected.
Pharmacist for how much topical steroid to use and where β steroid phobia causes widespread under-treatment.
Mental health where acne or eczema is affecting mood, sleep or school.
β οΈ What goes wrongComplications
Complications
Bacterial infection through breaks in the skin from scratching β monitor for infection, keep nails short, cleanse with mild soap and water, give antipruritics and antibiotics, and teach avoidance of offending agents
Psychosocial impairment from acne with stress, lowered self-esteem, and depression over appearance β monitor mood and behavior, encourage the adolescent to talk about feelings, support adherence, and have caregivers report mood changes
Skin scarring from scratching or delayed acne treatment, either atrophic with tissue loss or hypertrophic such as keloids β managed with topical medications, injectable steroids, silicone dressings, punch excision, cryosurgery, dermabrasion, or microdermabrasion, with realistic reassurance that therapy improves appearance
ATI Active Learning Template β System DisorderImmunizations
Filled from ATI chapter 34, row by row from that chapterβs own sections β 12 of 12 rows have content.
π§ What it isAlterations in Health (Diagnosis) Β· Health Promotion & Disease Prevention
Alterations in Health (Diagnosis)
Vaccines stimulate antibody production against a specific disease using a killed or weakened form of the organism. The Advisory Committee on Immunization Practices sets the schedule. Preterm infants receive each vaccine at the same chronologic age as full-term infants. A catch-up schedule on the CDC website covers missed doses. Goals are to reduce or eliminate infectious diseases in the population and to prevent those diseases and their complications.
Health Promotion & Disease Prevention
Hepatitis B: minimum age birth, 3 doses at birth, 1-2 months, and 6-18 months; at least 4 weeks between doses 1 and 2 and 8 weeks between doses 2 and 3; final dose no earlier than 24 weeks of age and at least 16 weeks after the first; withhold for preterm infants under 2,000 g if the mother is hepatitis B negative
Rotavirus: minimum age 6 weeks, 2 doses of Rotarix (2 and 4 months) or 3 doses of RotaTeq (2, 4, 6 months); do not start the series at 15 weeks0 days or older and give the last dose by 8 months0 days
DTaP: minimum age 6 weeks, 5 doses at 2, 4, 6 months, 15-18 months, and 4-6 years; at least 6 months between doses 3 and 4; dose 4 may be given as early as 12 months; dose 5 is unnecessary if dose 4 was given at 4 years or older and at least 6 months after dose 3
Tdap: minimum age 11 years, one dose at 11-12 years then Td booster every 10 years; one dose in each pregnancy between 27 and 36 weeks regardless of prior timing; for wounds, give Tdap or Td if a minor clean wound and over 10 years since the last dose, or over 5 years for all other wounds
Hib: minimum age 6 weeks, 4 doses (ActHIB, Pentacel, Vaxelis) at 2, 4, 6 months and a 12-15 month booster, or 3 doses (PedvaxHIB); use a different Hib product for the booster; only 1 dose is needed for unimmunized children 15 months or older
PCV13: minimum age 6 weeks, 4 doses at 2, 4, 6, and 12-15 months; follow current dual-series guidance with PPSV23 for high-risk conditions
π How it shows upAssessment β Risk Factors Β· Assessment β Expected Findings
Assessment β Risk Factors
From this module β built from the notes above on this page, not a section of the ATI chapter.
True contraindication: anaphylaxis to a previous dose or to a vaccine component.
Live vaccines (MMR, varicella, rotavirus, live influenza) are avoided in significant immunosuppression and in pregnancy.
Moderate or severe acute illness defers a dose; a mild cold with or without low fever does not.
Recent blood product or immunoglobulin can blunt the response to a live vaccine.
Egg allergy is not a contraindication to routine influenza vaccination, and a family history of reaction is not one either.
Assessment β Expected Findings
From this module β built from the notes above on this page, not a section of the ATI chapter.
Expected after a vaccine: low-grade fever, fussiness, and soreness, redness or a small lump at the site, for 1β2 days.
A small nodule at the injection site can persist for weeks and is harmless.
MMR and varicella may cause a mild rash or fever 7β12 days later, not immediately β parents need warning or they think it is unrelated illness.
Not expected: high fever, widespread hives, wheeze, facial or tongue swelling, or collapse. That is anaphylaxis.
π§ͺ How it is confirmedLaboratory Tests Β· Diagnostic Procedures
Laboratory Tests
From this module β built from the notes above on this page, not a section of the ATI chapter.
No routine bloodwork is needed before immunizing.
Titres may be checked to confirm immunity β rubella, varicella, hepatitis B β in health-care students and staff.
Hepatitis B surface antibody after the series in an infant of a positive mother.
Immune function testing before live vaccines where immunodeficiency is suspected.
Diagnostic Procedures
From this module β built from the notes above on this page, not a section of the ATI chapter.
None β immunization is a clinical decision from history and the schedule.
Review the record and use the catch-up schedule where doses are missing.
A lapsed schedule is resumed, never restarted.
Check the minimum interval between doses; too-early doses do not count.
Give the vaccine information statement and review it with guardians and older children, documenting each VIS publication date
Reassure caregivers that MMR is not associated with autism
IM route: vastus lateralis for infants and young children, deltoid for older children and adolescents
Subcutaneous route: outer upper arm or anterolateral thigh
Choose needle size by route, site, age, and volume β adequate length reduces injection site swelling and tenderness
Use comfort strategies to minimize discomfort
Effectiveness is shown by development of immunity and by local reactions resolving without pain, fever, or swelling
Anaphylaxis to any vaccine contraindicates further doses of that vaccine or any vaccine containing that substance
Medications
From this module β built from the notes above on this page, not a section of the ATI chapter.
Site by age:vastus lateralis for infants and toddlers, deltoid once the muscle is big enough, usually from about 3 years.
Needle length and gauge by age and muscle mass; 25 mm is common for infants.
Different vaccines go in different sites, or at least 1 inch apart, and are documented separately.
Epinephrine must be immediately available whenever vaccines are given.
Never mix vaccines in one syringe unless the product is licensed as a combination.
Therapeutic Procedures
From this module β built from the notes above on this page, not a section of the ATI chapter.
Comfort measures reduce distress and improve return rates: breastfeeding, sucrose, skin-to-skin for infants.
Hold the child securely with a parentβs help; give the injection quickly.
Observe for 15 minutes afterwards.
Older children: distraction, topical anesthetic, sitting up rather than lying down.
Give the most painful injection last.
π¬ Around the patientClient Education Β· Interprofessional Care
Client Education
From this module β built from the notes above on this page, not a section of the ATI chapter.
Give the Vaccine Information Statement before each vaccine β this is a legal requirement, not a courtesy.
Expect soreness and low fever; a cool compress and weight-appropriate acetaminophen or ibuprofen if needed.
Do not give an antipyretic prophylactically before the vaccine β it can reduce the immune response.
Return immediately for difficulty breathing, facial swelling, hives, or a child who becomes limp or unresponsive.
Keep the record and bring it to every visit; a lost record often means repeated doses.
Interprofessional Care
From this module β built from the notes above on this page, not a section of the ATI chapter.
Provider for the schedule and for judging true contraindications.
Pharmacist for storage and the cold chain β a break in it wastes the whole batch.
Public health for outbreak response, reporting and catch-up campaigns.
School nurse for entry requirements and exemptions.
Report significant adverse events through the national reporting system.
β οΈ What goes wrongComplications
Complications
DTaP mild: injection site redness, swelling, and tenderness, poor appetite, vomiting, low fever, drowsiness, irritability, anorexia
DTaP moderate reactions: crying that cannot be consoled lasting 3 hr or longer, temperature reaching 40.6 C (105 F) or above, seizures with or without fever, and a shock-like state
DTaP severe: acute encephalopathy, rare; encephalopathy within 7 days of a prior dose contraindicates further doses
LAIV: allergic reaction, vomiting or diarrhea, nasal congestion and runny nose
ATI Active Learning Template β System DisorderCommunicable Diseases
Filled from ATI chapter 35, row by row from that chapterβs own sections β 12 of 12 rows have content.
π§ What it isAlterations in Health (Diagnosis) Β· Health Promotion & Disease Prevention
Alterations in Health (Diagnosis)
Communicable diseases spread by airborne, droplet, or direct contact transmission, and most are vaccine-preventable. Antibiotics and antitoxins reduce serious complications. Primary prevention means immunization; secondary prevention means limiting spread to others. Immunization is the single best method of prevention.
Health Promotion & Disease Prevention
Conjunctivitis spreads by direct contact; viral requires contact precautions and bacterial requires standard precautions
Viral conjunctivitis follows a viral infection, starts in one eye and spreads to the other, and clears on its own in 7-14 days; bacterial clears with topical antibiotics; allergic clears with allergy medication
Infectious mononucleosis (Epstein-Barr virus) spreads in oral secretions, incubation 3-6 weeks; healthy carriers shed the virus in saliva for life and people with mono transmit for weeks
Fifth disease spreads through respiratory secretions and blood product transfusion, incubation 4-14 days and up to 21, most contagious in the 5 days before the rash, droplet precautions
Mumps spreads by direct contact with respiratory droplets, incubation 14-21 days, contagious 1-2 days before symptoms until 5 days after they begin
Pertussis spreads by direct or indirect contact with respiratory secretions and freshly contaminated articles, incubation 1-3 weeks, most contagious during the catarrhal stage before paroxysms begin
π How it shows upAssessment β Risk Factors Β· Assessment β Expected Findings
Assessment β Risk Factors
Prior communicable disease history and recent exposure to a known case
Not immunized or behind on immunizations
Immunocompromised status and chronic illness
Crowded living conditions, poor sanitation, poor nutrition
Poor oxygenation and impaired circulation
Assessment β Expected Findings
Conjunctivitis: pink or red sclera, swollen conjunctiva, excessive tearing; bacterial adds yellow-green purulent discharge and morning eyelid crusting, viral produces watery discharge
Fifth disease prodrome: several days of fever, runny nose, headache
Fifth disease rash lasting 7 days to several weeks: red slapped-cheek facial rash on days 1-4, then symmetric maculopapular red spots spreading proximal to distal on the extremities over a week, with a secondary itchy rash possible elsewhere including the soles
Measles prodrome 3-4 days before the rash: mild to moderate fever, cough, runny nose, red eyes, sore throat
Measles: Koplik spots (tiny white spots) in the mouth 2 days before the rash, then a red or reddish-brown rash starting on the face and moving downward with a fever spike
Pertussis in infants and children: seizures, apnea, pneumonia, ear infection, encephalopathy, hemorrhage, weight loss, hernia, and death; in teens and adults: rib fracture, syncope, pneumonia, weight loss, and loss of bladder control
Rubella: complications are generally rare, but maternal infection during pregnancy causes fetal birth defects including deafness, heart defects, cognitive deficits, and liver and spleen damage
Rubeola: ear infection, pneumonia, encephalitis, laryngitis, death
ATI Active Learning Template β System DisorderAcute Otitis Media
Filled from ATI chapter 36, row by row from that chapterβs own sections β 12 of 12 rows have content.
π§ What it isAlterations in Health (Diagnosis) Β· Health Promotion & Disease Prevention
Alterations in Health (Diagnosis)
Acute otitis media is infection of the middle ear structures and one of the most common acute childhood illnesses. Otitis media with effusion is fluid in the middle ear without infection. Most cases stem from eustachian tube dysfunction and many clear spontaneously within days, but repeated infections can impair hearing and speech.
Health Promotion & Disease Prevention
From this module β built from the notes above on this page, not a section of the ATI chapter.
Pneumococcal and influenza vaccination reduce episodes.
Breastfeeding is protective; avoid bottle propping and feeding lying flat.
Eliminate tobacco smoke exposure β one of the strongest modifiable risk factors.
Explain the anatomy: a childβs eustachian tube is shorter, wider and more horizontal, which is why this is a childhood disease.
Limit pacifier use after 6 months.
π How it shows upAssessment β Risk Factors Β· Assessment β Expected Findings
Assessment β Risk Factors
Children's eustachian tubes are shorter and more horizontal than adults'; incidence peaks in the first 24 months and declines after age 5
Tugging or rubbing the ear and rolling the head side to side
Ear pain, loss of appetite, nausea, vomiting, fever, lethargy
Acute otitis media: bulging inflamed tympanic membrane, purulent material in the middle ear or draining from the canal, decreased or absent movement on pneumatic otoscopy, head and neck lymphadenopathy
Chronic disease brings hearing difficulty and speech delay
Otitis media with effusion: feeling of ear fullness, opaque tympanic membrane with decreased movement, transient hearing loss
π§ͺ How it is confirmedLaboratory Tests Β· Diagnostic Procedures
Laboratory Tests
From this module β built from the notes above on this page, not a section of the ATI chapter.
None routinely β the diagnosis is made by looking at the drum.
Culture of middle ear fluid only if tympanocentesis is performed for treatment failure.
Blood cultures if the child is systemically unwell or very young.
Hearing assessment after recurrent episodes or persistent effusion.
Diagnostic Procedures
Pneumatic otoscope lets the provider visualize the tympanic membrane and middle ear and assess membrane movement
Pull the pinna down and back under 3 years, up and back over 3 years
Provide comfort measures and diversional activities
Position the child upright
Manage fever
Medications
Acetaminophen or ibuprofen for analgesia and fever
Antibiotics: amoxicillin, amoxicillin-clavulanate, or azithromycin orally for 10 days, or IM ceftriaxone for three doses
Antibiotics are recommended over 6 months of age for severe symptoms β increased pain or temperature above 39 C (102.2 F) β lasting more than 2 days, and for children 6-23 months with bilateral disease
Benzocaine ear drops relieve pain
Therapeutic Procedures
Management depends on physical findings, age, and history
AAP recommends watchful waiting for 2-3 days before antibiotics in children 2-12 years with uncomplicated, nonrecurrent disease
Myringotomy with tympanostomy tube placement for repeated episodes, done as outpatient surgery under general anesthesia, sometimes by laser
A small incision in the tympanic membrane allows tiny plastic tubes to equalize pressure and reduce effusion; postoperative pain is uncommon and mild if present
π¬ Around the patientClient Education Β· Interprofessional Care
Client Education
Finish the entire antibiotic course and watch for allergy such as rash or difficulty breathing
Feed the child upright when bottle- or breastfeeding
Apply antibiotic ear drops as prescribed and use comfort measures
Limit exposure to secondhand smoke and to people with respiratory infections
Seek care at the first sign of infection such as behavior change or ear tugging
Keep immunizations current
Interprofessional Care
From this module β built from the notes above on this page, not a section of the ATI chapter.
Provider for diagnosis and the decision to treat or observe.
ENT surgeon for recurrent infection or persistent effusion β tympanostomy tubes.
Audiology to check hearing after repeated episodes.
Speech and language therapy where hearing loss has delayed speech.
β οΈ What goes wrongComplications
Complications
Hearing loss and speech delay β assess and monitor for deficits, refer for audiology testing, and arrange speech therapy if needed
📋 HIV/AIDS6 parts
ATI Active Learning Template β System DisorderHIV/AIDS
Filled from ATI chapter 37, row by row from that chapterβs own sections β 12 of 12 rows have content.
π§ What it isAlterations in Health (Diagnosis) Β· Health Promotion & Disease Prevention
Alterations in Health (Diagnosis)
HIV infects CD4 T lymphocytes, impairing and destroying immune cells and progressively weakening immune function. Adolescents 13-20 years are managed following adult HIV/AIDS content.
Health Promotion & Disease Prevention
From this module β built from the notes above on this page, not a section of the ATI chapter.
Perinatal transmission is largely preventable β maternal antiretroviral therapy, appropriate delivery, infant prophylaxis and avoiding breastfeeding where formula is safe.
Adolescents: condoms, testing, and pre-exposure prophylaxis where indicated.
Full vaccination including annual influenza; live vaccines depend on the CD4 count.
Prophylaxis against opportunistic infection per CD4 threshold.
Address stigma and disclosure directly β it drives whether the child takes their medicines.
π How it shows upAssessment β Risk Factors Β· Assessment β Expected Findings
Assessment β Risk Factors
Perinatal transmission or ingestion of breast milk from a person with HIV
Exposure to blood products or body fluids containing the virus
Sexual assault
Risky behaviors such as unprotected sex and IV substance use
Existing sexually transmitted infections
Lack of awareness of risk factors and transmission routes
Assessment β Expected Findings
Immunologic staging by CD4 count under 12 months: stage 1 is 1,500 cells/microliter or more, stage 2 is 750-1,499, stage 3 is under 750
Ages 1-5 years: stage 1 is 1,000 or more, stage 2 is 500-999, stage 3 is under 500
Age 6 and older: stage 1 is 500 or more, stage 2 is 200-499, stage 3 is under 200
Clinical category N is not symptomatic β no HIV-attributable findings or only one mildly symptomatic condition
Category A is mildly symptomatic, including recurrent sinusitis
Severely symptomatic disease includes opportunistic infections such as pulmonary candidiasis
π§ͺ How it is confirmedLaboratory Tests Β· Diagnostic Procedures
Laboratory Tests
From this module β built from the notes above on this page, not a section of the ATI chapter.
Infants of positive mothers need virological testing (PCR), not antibody testing β maternal antibody persists to about 18 months and gives a false positive.
CD4 count for immune status and viral load for treatment response.
CBC, renal and liver function for drug toxicity.
Resistance testing where the viral load fails to suppress.
Screening for TB, hepatitis B and C, and other STIs in adolescents.
Diagnostic Procedures
From this module β built from the notes above on this page, not a section of the ATI chapter.
18 months and older: positive ELISA confirmed by Western blot
Under 18 months born to an infected parent: positive polymerase chain reaction test
Rapid saliva testing detects antibodies noninvasively but a positive result requires blood confirmation
Non-nucleoside reverse transcriptase inhibitors β delavirdine, efavirenz, nevirapine, doravirine, etravirine, rilpivirine β bind viral DNA for direct inhibition
Trimethoprim-sulfamethoxazole prophylaxis for every infant born to an infected parent until HIV infection is ruled out
Therapeutic Procedures
From this module β built from the notes above on this page, not a section of the ATI chapter.
Combination antiretroviral therapy, started early and taken without gaps.
Adherence is everything β missed doses breed resistance. Ask how many were missed this week, not whether they take it.
Nutritional support and growth monitoring.
Age-appropriate disclosure to the child, planned with the family rather than improvised.
Transition planning to adult services during adolescence, started early.
π¬ Around the patientClient Education Β· Interprofessional Care
Client Education
Take medications on a regular schedule without missed doses; the illness is chronic and treatment is lifelong
Watch for adverse effects and use strategies to lessen them
Call the provider for headache, fever, lethargy, warmth, tenderness or redness at joints, or neck stiffness
Practice safe handling of needles and syringes
Discuss transmission routes with adolescents including sexual transmission and IV substance use, and confirm they understand safer sex practices
Interprofessional Care
Social services for access to health care and medications
Dietitian for nutritional support
β οΈ What goes wrongComplications
Complications
From this module β built from the notes above on this page, not a section of the ATI chapter.
Failure to thrive β obtain baseline height and weight and monitor, promote optimal nutrition including total parenteral nutrition if needed, assess growth and development for delays, and provide age-appropriate toys and peer play
Pneumocystis pneumonia β monitor respiratory rate and effort, oxygen saturation, and breath sounds, give antibiotics, antipyretics, and analgesics, maintain hydration and fluid and electrolyte balance, promote rest, and teach infection prevention and medication adherence
📋 Burns6 parts
ATI Active Learning Template β System DisorderBurns
Filled from ATI chapter 41, row by row from that chapterβs own sections β 12 of 12 rows have content.
π§ What it isAlterations in Health (Diagnosis) Β· Health Promotion & Disease Prevention
Alterations in Health (Diagnosis)
Thermal, cold, chemical, electrical, and radioactive agents destroy skin layers and underlying tissue. Thermal burns follow exposure to flame, hot surfaces, or hot liquids. Chemical burns follow contact with acids, alkalis, or organic compounds, including household and industrial cleaners. Electrical burns occur when current passes through the body and can destroy tissue, cost organ function, and require amputation. Burn depth is graded superficial, superficial partial thickness, deep partial thickness, full thickness, and deep tissue extension.
Health Promotion & Disease Prevention
Provide adequate supervision and establish a safe play area
Store hot liquids, electrical cords, and anything dangling where a child cannot reach
π How it shows upAssessment β Risk Factors Β· Assessment β Expected Findings
Assessment β Risk Factors
Abuse or neglect
Inadequate supervision
Assessment β Expected Findings
Determine which agent burned the child (electrical, chemical, moist heat, dry heat, ionizing radiation), how long contact lasted, and which body area was involved
Younger children sustain deeper injuries because their skin is thinner
Extent measured as total body surface area using age-related charts and expressed as a percentage
Severity depends on age, causative agent, body area, and burn extent and depth
American Burn Association grading β minor: partial thickness under 10% TBSA, usually outpatient; moderate: partial thickness 10-15% TBSA and full thickness 2-10% TBSA, treated at a hospital with burn expertise; major: partial thickness over 20% TBSA, requiring a burn center
π§ͺ How it is confirmedLaboratory Tests Β· Diagnostic Procedures
Laboratory Tests
Major burns: CBC, electrolytes, BUN, ABGs, random glucose, liver enzymes, urinalysis
Diagnostic Procedures
From this module β built from the notes above on this page, not a section of the ATI chapter.
Estimate extent with a pediatric chart β the adult rule of nines is wrong in children, whose heads are proportionally much larger.
Assess depth: superficial, superficial partial, deep partial, full thickness.
Look for airway involvement β facial burns, singed nasal hair, soot, hoarseness, stridor. Intubate early rather than late.
Weigh, because fluid resuscitation and every drug depend on it.
Consider non-accidental injury: sharply demarcated immersion lines, a stocking-glove distribution, or a history that does not fit.
On-site: stop the burning process and remove the child from the source
Lay the child flat and smother flames by rolling them in a rug or blanket, or roll the child slowly if nothing is available
Remove clothing and jewelry that conduct heat
Apply tepid water soaks or run water over the injury β never ice, which risks hypothermia
Flush liquid chemical burns with a large volume of water for extended cooling
Assess airway patency, cover the burn with a clean dry cloth to prevent contamination, and transport for evaluation
Phases of management: acute/emergent-resuscitative focused on shock and respiratory complications, management focused on preventing infection and closing the wound, and rehabilitative focused on preventing scarring complications after wound closure
Airway is the priority β give humidified 100% oxygen as prescribed, monitor respirations and oxygen saturation, check ABGs and carbon monoxide level
Prepare to intubate if stridor, nasal flaring, drooling, or respiratory distress appear, and prepare for escharotomy with full-thickness chest burns to allow ventilation
Medications
Topical agents for partial-thickness burns β silver sulfadiazine (inhibits bacterial growth), mafenide acetate, collagenase (santyl), bacitracin β applied to a cleansed, debrided area with sterile gloves
Topical prepackaged mesh gauze dressings protect the wound, improve comfort, speed healing, and reduce dressing change frequency
Morphine sulfate IV for analgesia, sometimes premedicated before dressing changes β monitor for respiratory depression and discuss dependency risk with prolonged use
Midazolam, fentanyl, propofol, and nitrous oxide for sedation and analgesia given IV just before a procedure
Nitrous oxide and oxygen mixture is a short-acting analgesic gas that relieves anxiety and raises the pain threshold during procedures
Therapeutic Procedures
Open method leaves the wound uncovered with a topical antimicrobial
Modified method applies antimicrobial to thin gauze or net secured over the wound
Occlusive method impregnates gauze with antimicrobial or applies it directly, then layers gauze secured with gauze or net
Temporary skin coverings promote healing of large partial-thickness burns and need repeated surgical application
Allograft (homograft) from human skin covers partial and full thickness wounds, with rejection possible 14 days after application
Xenograft from porcine skin covers partial-thickness wounds but does not vascularize, so it must be changed every few days
π¬ Around the patientClient Education Β· Interprofessional Care
Client Education
Continue range-of-motion exercises and physical therapy to prevent contractures
Assess the wound for infection and perform wound care at home
Home safety: cover electrical outlets, supervise bathing, keep irons out of reach, and teach the danger of playing with matches
Avoid sun between 1000 and 1400, wear protective clothing, and use sunscreen
Growth and weight delays are possible for up to 3 years after a burn, and bone remodeling risk is increased
Arrange interprofessional referrals for care after discharge
Interprofessional Care
Refer to nutrition, social support, respiratory therapy, occupational and physical therapy, and individual and family counseling as prescribed
β οΈ What goes wrongComplications
Complications
Direct thermal injury with face and lip burns damages the tracheobronchial tree after inhaling heated gases and toxic combustion products, and can be delayed 24-72 hr β wheezing, increased secretions, hoarseness, wet rales, singed nasal hairs, laryngeal edema, carbonaceous secretions
Carbon monoxide injury occurs in enclosed spaces, causing mental status change and dyspnea β maintain airway and ventilation with 100% oxygen
Pulmonary injury from inhaled fumes, vapors, gases, and smoke β maintain the airway with intubation or sometimes tracheostomy and give oxygen
Hypertrophic scarring from collagen deposition during healing appears raised β apply uniform pressure, fit custom pressure garments, continue physical therapy, give cetirizine or diphenhydramine for itching, and massage and moisturize scars as prescribed
Wound sepsis, since burn wounds are a growth medium for bacteria β disorientation is an early sign, followed by spiking fever, decreased bowel sounds, tachycardia, tachypnea, thrombocytopenia, oliguria, and leukopenia
π Notes & key concepts
The lines from this module that carry a number, a dose or an absolute rule β the ones that decide questions. Everything else is on the cards above.
HIV: CD4 tracks disease, <200 defines category C/AIDS. PCR/viral culture diagnoses infants; ELISA plus Western blot after 18 months. Standard precautions only. Live vaccines need caution.
Anaphylaxis: IM epinephrine is the first action.
Contagion endpoints: varicella until every lesion crusts; rubeola 4 days after rash onset; fifth disease is no longer contagious once the rash appears.
Mono: splenomegaly means no contact sports for about 4 weeks. Monospot positive. Recovery takes months.
Impetigo: honey-colored crusts, mupirocin. Head lice: 1% permethrin plus nit combing, repeat in 2 weeks. Pinworms: tape test, mebendazole for the whole household.
Burns: airway first, then fluids, then wound care and pain. Tepid soaks, never ice. Do not drain blisters. Children's thinner skin means deeper injury for the same exposure.
Diaper dermatitis: zinc barrier and air exposure. Never talc.
π― Module quiz
Questions for this module. They also feed the Mega Quiz.