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Final · Week 13 · BIO 280 Pathophysiology

M13 · Gastrointestinal Disorders

From mouth to colon: reflux, ulcers, obstruction and the two inflammatory bowel diseases that get confused.

🧩 5 study cards⭐ exam spotlight📊 3 comparison tables🚨 3 never-do rules📱 Foldy-friendly
M13Gastrointestinal DisordersWeek 13
📚 Outline: Module 13 — Part 1 Oral to Esophageal · Part 2 Gastric · Part 3 Lower GI
💡 The one idea

The gut is a tube with a protective lining and a one-way valve system. Almost every disorder here is acid where it should not be, a blockage, or inflammation of the wall.

Where the pain is, and what it does after eating, usually names the diagnosis.

👄 Mouth to esophagus
  • GERD — a lax lower esophageal sphincter lets acid back up. Heartburn worse lying flat or after meals, regurgitation, sour taste, chronic cough, hoarseness. Aggravated by caffeine, alcohol, chocolate, fat, peppermint, nicotine, and large late meals.
  • Barrett esophagus — chronic acid causes metaplasia of the lining. It is pre-malignant and needs surveillance.
  • Hiatal hernia — stomach pushes up through the diaphragm; worsens reflux.
  • Achalasia — the sphincter fails to relax; food sticks. Dysphagia to solids and liquids from the start.
  • Esophageal varices — dilated veins from portal hypertension. Painless, catastrophic bleeding.

Progressive dysphagia to solids first, then liquids, with weight loss, suggests an obstructing tumor rather than a motility problem. That direction matters.

🚨 Peptic ulcer disease — gastric vs duodenal
Gastric ulcerDuodenal ulcer
Pain vs food Worse with eating — 30–60 min after Better with eating, then returns 2–3 h later; wakes them at night
WeightLoss — eating hurtsGain or stable
AcidNormal or low; the defense has failed High
Malignant?Can beRarely

H. pylori and NSAIDs are the two big causes. H. pylori weakens the mucus barrier; NSAIDs block the prostaglandins that maintain it. Both let acid reach the wall.

Sudden severe abdominal pain with a rigid, board-like abdomen means perforation. Coffee-ground vomit or melaena means bleeding. Both are surgical emergencies.

🪥 Crohn vs ulcerative colitis
Crohn diseaseUlcerative colitis
WhereMouth to anus, anywhere — usually terminal ileumColon and rectum only, continuous from the rectum up
PatternSkip lesions — patchy Continuous, no gaps
DepthFull thickness — hence fistulas, strictures, abscessesMucosa and submucosa only
StoolDiarrhea, often without visible blood; steatorrhoea Bloody diarrhea with mucus, urgency, tenesmus
NutritionMalabsorption — B12, iron, fat Less affected
Big riskObstruction, fistula Toxic megacolon; higher colon cancer risk

Crohn is patchy, deep and anywhere. UC is continuous, shallow and colon only.

⭐ Lower GI — obstruction and the acute abdomen
Small bowel obstructionLarge bowel obstruction
VomitingEarly and profuseLate, may be faeculent
DistensionLessMarked
PainCramping, frequent wavesCramping, less frequent
Commonest causeAdhesions, hernia Tumor, volvulus, diverticular disease
  • Appendicitis — periumbilical pain migrating to the right lower quadrant (McBurney point), rebound tenderness, anorexia, low fever.
  • Diverticulitisleft lower quadrant pain, fever, altered bowel habit. Diverticulosis is the pouches; diverticulitis is when they inflame.
  • Peritonitis — rigid abdomen, rebound tenderness, absent bowel sounds, shallow breathing, lying still.
  • C. difficile — follows antibiotics; profuse watery diarrhea. Contact precautions and soap-and-water handwashing — alcohol gel does not kill the spores.

A patient with abdominal pain who is lying completely still has peritoneal irritation. One who is writhing and cannot get comfortable has colic — obstruction or a stone. That difference is diagnostic.

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