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Exam 3 · Week 6 · BIO 280 Pathophysiology

M6 · Musculoskeletal · Skin · Burns

Bone, muscle and skin: what breaks, what erodes, and why a major burn is a fluid problem before it is a skin problem.

🧩 7 study cards⭐ exam spotlight📊 3 comparison tables🚨 3 never-do rules📱 Foldy-friendly
M6Musculoskeletal · Skin · BurnsWeek 6
📚 Outline: Module 6 — Part 1 Musculoskeletal Disorders · Part 2 Skin Disorders · Part 3 Burns
💡 The one idea

Skin is an organ, and losing it is a systemic event. A large burn kills through fluid shift, heat loss and infection long before the wound itself matters.

First 24 hours = fluid. After 48 hours = infection.

🦴 Bone — osteoporosis vs osteoarthritis vs osteomyelitis
What it isKey point
OsteoporosisBone resorption outruns formation — less bone, normal composition Silent until it fractures. Post-menopausal estrogen loss is the biggest driver. Vertebral, hip and wrist.
OsteomalaciaVitamin D deficiency — bone is soft, not thinRickets in children; bone pain and bowing
OsteoarthritisCartilage wear Asymmetric, weight-bearing, <30 min stiffness, worse with use
OsteomyelitisBone infection, usually S. aureus Bone has poor blood supply — needs weeks of IV antibiotics
GoutUric acid crystals in a joint Sudden, exquisitely painful, classically the great toe at night
🚨 Compartment syndrome & fat embolism

Pain out of proportion to the injury, unrelieved by opioids, and worse on passive stretch is compartment syndrome until proven otherwise. Do not elevate the limb above the heart and do not apply ice — both reduce perfusion further.

The six Ps: Pain (first and most reliable), Pressure, Paresthesia, Pallor, Paralysis, Pulselessness. Pulselessness is last — waiting for it means waiting until the muscle is dead.

Fat embolism follows long-bone and pelvic fractures, usually within 24–72 h: the triad is hypoxia, confusion and a petechial rash over the chest and axillae.

🩹 Skin — pressure injuries
StageWhat you see
1Intact skin, non-blanchable redness
2Partial thickness — shallow open ulcer or an intact/ruptured blister. Dermis is visible. No slough.
3Full thickness — subcutaneous fat visible; slough may be present; bone/tendon/muscle not exposed
4Full thickness — bone, tendon or muscle exposed
UnstageableBase obscured by slough or eschar — you cannot stage what you cannot see
Deep tissuePersistent maroon or purple discoloration of intact skin

Pressure, shear, friction and moisture are the four forces. Bony prominences are where they meet: sacrum, heels, ischium, trochanters, occiput.

⭐ Burn depth
DepthLayersLooks likePain
SuperficialEpidermisRed, dry, blanchesPainful
Partial thicknessInto dermis Blisters, moist, weepingMOST painful
Full thicknessThrough dermis White, leathery, waxy, dry Painless — nerves destroyed
Deep full thicknessInto muscle and bone Charred, blackenedPainless

A patient who cannot feel the burn has destroyed nerve endings. Painless is worse, never better.

💧 Rule of Nines and Parkland
  • Head & neck 9% · Each arm 9% · Each leg 18%
  • Front torso 18% · Back torso 18% · Perineum 1%

Children have proportionally bigger heads, so pediatric charts shift percentage from the legs to the head.

4 mL × kg × %TBSA Lactated Ringer's over 24 hours, timed from the moment of the burn, not from arrival. Half in the first 8 hours, the other half over the next 16.

70 kg, 50% TBSA: 4 × 70 × 50 = 14,000 mL7,000 mL in 8 h (~875 mL/h), then 7,000 mL over 16 h (~437 mL/h).

The best indicator that fluids are working is urine output — adults 30–50 mL/hr, children 1 mL/kg/hr. Not blood pressure, not heart rate.

🔥 Airway comes before all of it

Suspect inhalation injury with facial burns, singed nasal hair, soot in the mouth, hoarseness, stridor or a fire in an enclosed space. The airway swells over hours — intubate early, because after the swelling you cannot.

Circumferential burns of the chest or a limb act like a tourniquet as edema builds — they need escharotomy. Carbon monoxide poisoning gives a normal pulse oximetry reading with a hypoxic patient: treat with 100% oxygen.

🎯 Module quiz

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