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Exam 4 · Week 8 · BIO 280 Pathophysiology

M8 · Endocrinology & Endocrine Disorders

Feedback loops, and the six gland pairs where too much and too little are mirror images of each other.

🧩 6 study cards⭐ exam spotlight📊 4 comparison tables🚨 2 never-do rules📱 Foldy-friendly
M8Endocrinology & Endocrine DisordersWeek 8
📚 Outline: Module 8 — Part 1 Endocrinology · Part 3 Endocrine Disorders
💡 The one idea

Nearly every endocrine disease is one gland doing too much or too little, and the two are mirror images. Learn one side properly and you get the other for free.

Negative feedback: the product switches off its own signal. If the product is high and the signal is also high, the problem is the gland giving the signal.

🔄 How the axis works

Hypothalamus → pituitary → target gland → hormone. The hormone then feeds back and turns the top of the chain off.

  • Primary problem = the target gland itself. Hormone low, and the pituitary signal (TSH, ACTH) is high because it is shouting at a gland that cannot answer.
  • Secondary problem = the pituitary. Hormone low and the signal is low too.

Both numbers low → the problem is upstream. One low, one high → the problem is the gland.

🧘 Thyroid — the whole body's thermostat
HypothyroidHyperthyroid
Everything isSlow and cold Fast and hot
SignsFatigue, weight gain, cold intolerance, constipation, dry skin, hair loss, bradycardia, depression, menorrhagia Weight loss with good appetite, heat intolerance, diarrhea, tremor, tachycardia, anxiety, insomnia, exophthalmos in Graves
TSH (primary)HighLow
T3 / T4LowHigh
CrisisMyxedema coma — hypothermia, hypotension, bradycardia, unresponsive Thyroid storm — fever >38.5, tachycardia, agitation, delirium

TSH moves opposite to the thyroid hormone in primary disease. That inverse is the single most tested relationship in the module.

🦵 Parathyroid and calcium

PTH raises serum calcium — it pulls it from bone, reabsorbs it in the kidney, and activates vitamin D to absorb it from the gut. Phosphate moves the opposite way.

  • Hyperparathyroid → high calcium → stones, bones, groans and moans: renal calculi, bone pain and fractures, constipation and abdominal pain, lethargy and confusion. Reflexes are depressed.
  • Hypoparathyroid → low calcium → irritable everything: tetany, perioral tingling, muscle cramps, Chvostek and Trousseau signs, and the one that kills — laryngospasm. Reflexes are brisk.

After thyroid or parathyroid surgery, hoarseness, tingling around the mouth or stridor means the calcium has crashed. Keep calcium gluconate available.

🚨 Adrenal — Cushing vs Addison
Cushing (too much cortisol)Addison (too little)
BodyCentral obesity, moon face, buffalo hump, thin limbs, purple striae, thin skin, bruising Weight loss, fatigue, anorexia, bronze hyperpigmentation
Sodium / waterRetained — hypertension, edemaLost — hypotension, dehydration
PotassiumLowHigh
GlucoseHighLow
ImmunitySuppressed — infection risk, poor healing

Addisonian crisis — profound hypotension, hyperkalemia, hypoglycemia and shock, often triggered by stress, infection, or abruptly stopping steroids. Long-term corticosteroids must always be tapered, never stopped suddenly.

⭐ Diabetes — and the two emergencies
Type 1Type 2
ProblemNo insulin — autoimmune beta-cell destructionInsulin resistance plus relative deficiency
OnsetUsually young, abruptUsually adult, gradual
KetonesYes — DKA Rarely — HHS instead

The three Ps: polyuria, polydipsia, polyphagia. Glucose spills into the urine and drags water with it.

DKAHHS
InsulinNoneA little — enough to block ketones
Glucose>250>600
pHAcidoticNormal
HallmarkKussmaul respirations, fruity breath, abdominal painProfound dehydration and neurological change
OnsetHoursDays

Treatment order for both: fluids first, then insulin — and check the potassium before the insulin. Acidosis has driven potassium out of the cells, so serum potassium can look normal while total body potassium is depleted; insulin drives it back in and the level can fall far enough to stop the heart. At a glucose of ~250, add dextrose and keep the insulin running — the insulin is there to switch off ketone production, not only to lower the number.

🎯 Module quiz

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