NUR 198 ยท Exam 3 ยท Cardiac Remediation

Cardiac Concepts — Stop Memorizing, Start Thinking

The five cardiac concepts and the thinking process that gets you to the answer.

What is wrong with the heart, is blood reaching the body, and what kills first?Electrical problem, pump problem, or vessel problem — name it before you look at the options.
Every highlighted answer was a blank in the handout.
Step 1 · Start Here 1 · CAD → MI 2 · Heart Failure 3 · Dysrhythmias 4 · Hypertension 5 · Shock The NCLEX Framework 10 Practice Questions The 5 Questions, Every Time โญ Must Not Forget

Step 1 · Every Cardiac Question Starts Here โญ

Before you look at the answer choices. Every time. Same three questions.

Ask 1What is wrong with the heart?
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Ask 2Is blood reaching the body?
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Ask 3What will kill first?

โœ… 1. What is wrong with the heart?

Type of problemWhat it meansClassic examples
โšก ElectricalThe signal is wrong — too fast, too slow, or disorganized. The muscle may be fine; the wiring is not.A-fib, VT, VF, bradycardia, heart block, asystole
๐Ÿ’ช PumpingThe muscle cannot move blood forward, so it backs up behind the failing side.Heart failure, cardiogenic shock, MI damage, pulmonary edema
๐Ÿฉธ Blood vesselThe pipes are narrowed, blocked, or under too much pressure, so tissue does not get oxygen.CAD, angina, MI, hypertension, PAD

โœ… 2. Is blood reaching the body?

Perfusion is the whole question. Everything else is detail.

โœ… 3. What will kill the patient first?

No oxygenAirway/breathing first — the brain dies in minutes without O₂.
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No perfusionBlood is not moving — organs start failing (brain, kidney, skin show it first).
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Dangerous rhythmVF or pulseless VT — no cardiac output at all. Defibrillate.

โค๏ธ 1 · Coronary Artery Disease → Myocardial Infarction ๐Ÿšจ

The problem is a blood vessel problem. Because the pipe narrows, the muscle behind it starves.

What happens?

Step 1Plaque narrows the artery
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Step 2Heart muscle gets less oxygen
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Step 3Chest pain (angina)
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Step 4Complete blockage = MI

Think NCLEX ๐Ÿง 

Chest pain + diaphoresis + nausea = think heart first.

Why does that combination mean heart? Diaphoresis and nausea are sympathetic and vagal responses to a dying myocardium — they mean the body is in distress, not just uncomfortable. Indigestion does not make you cold and sweaty.

Stable anginaUnstable anginaMI
TriggerExertion or stress — predictableAt rest, new, or worseningAny time, often at rest
Relieved by rest / nitroYesNot fullyNo
TroponinNormalNormalElevated — muscle is dying
MeaningDemand > supply, temporarilyWarning shot — treat as ACSTissue death. Time = muscle.

Never walk, feed, or delay a patient with active chest pain. Get the 12-lead ECG.

โค๏ธ 2 · Heart Failure

The problem is a pumping problem. Because the heart cannot move blood forward, it backs up behind the side that failed.

Left failsโžก๏ธ Fluid backs into the lungs
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You seeCrackles, dyspnea, orthopnea, cough, frothy/pink sputum, low SpO₂
Right failsโžก๏ธ Fluid backs into the body
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You seePeripheral edema, JVD, ascites, weight gain, enlarged liver

Remember

SideMemory hookBacks up intoHallmark finding
LeftL = LungsPulmonary circulationCrackles
RightR = Rest of the bodySystemic circulationPeripheral edema and JVD

Daily weight is the earliest sign. 1 kg gained โ‰ˆ 1 L of fluid retained. Teach her to report 2–3 lb in 24 hours or 5 lb in a week.

โค๏ธ 3 · Dysrhythmias

The problem is an electrical problem. Because the signal is disorganized, the pump may or may not still be filling and ejecting.

Ask yourself four questions

โœ” 1 Regular?Are the R–R intervals the same? Irregularly irregular → think A-fib.
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โœ” 2 Rate?<60 = brady, 60–100 = normal, >100 = tachy.
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โœ” 3 P waves?One upright P before every QRS? No P waves → not sinus.
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โœ” 4 QRS?Narrow = above the ventricles. Wide = ventricular → more dangerous.

Then ask…

Is this rhythm perfusing the patient? If not → priority.

Perfusing (stable)Not perfusing (unstable)
Looks likeAwake, talking, BP adequate, no chest pain, warm skinConfused or unresponsive, hypotensive, chest pain, cold clammy, low urine output
You doAssess, monitor, treat the cause, give the ordered drugTreat the rhythm now — electricity, CPR, or rapid escalation
No pulse at allVF or pulseless VT → CPR + defibrillate. Asystole/PEA → CPR + epinephrine, never shock asystole.

โค๏ธ 4 · Hypertension

The problem is a blood vessel problem that turns into a pump problem.

Step 1High pressure
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Step 2Heart works harder
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Step 3Heart gets thicker
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Step 4Eventually weak
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Step 5Heart failure

Don't memorize. Follow the chain. The heart is pushing against high afterload every beat, so the left ventricle hypertrophies. Thick muscle is stiff muscle — it fills poorly and outgrows its own blood supply, so it eventually fails.

Normal BP: less than 120/80 mm Hg. Hypertension: 130/80 mm Hg or higher on two separate readings. Hypertensive crisis: above 180/120 mm Hg — ๐Ÿšจ check for organ damage (chest pain, vision change, headache, neuro change).

Hypertension is silent. The damage is the diagnosis.

โค๏ธ 5 · Shock ๐Ÿšจ

Cardiogenic shock = the heart cannot pump. Everything else comes from that.

StartPump fails
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ThenCardiac output drops
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ThenTissues do not get oxygen
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ThenOrgans fail in order
SignWhy it happens
Low BPLess blood ejected per beat → less pressure in the system.
Fast HRCompensation — the body speeds up to make up for the small stroke volume.
Cold skinBlood is shunted away from the skin to protect heart and brain.
Poor urine outputKidneys are not being perfused. <30 mL/hr is the red flag.
ConfusionThe brain is not being perfused — often the earliest sign, and the one students skip.

New confusion in a cardiac patient is a perfusion emergency until proven otherwise.

The NCLEX Framework โญ

Every question. STOP. Then ask:

Practice Questions ๐Ÿง 

Run the same three questions on each one before you open the answer.

1. A client suddenly develops crushing chest pain that radiates to the left arm and is diaphoretic. What is the priority?
  1. Obtain a pain history
  2. Perform a 12-lead ECG
  3. Offer food
  4. Encourage ambulation

Break it down: โœ” Chest pain โœ” Diaphoresis → Could this be an MI? YES. Which action confirms it fastest?

Show the answer

B — Perform a 12-lead ECG. It is the fastest test that identifies ST elevation, and it drives every decision after it (cath lab vs. medical management). Standard is within 10 minutes of the complaint.

AA full pain history is assessment you can gather while the ECG runs; alone it delays diagnosis.
CKeep the client NPO. Food adds cardiac workload and blocks an urgent cath.
DAmbulating raises oxygen demand in a heart that already cannot meet demand. Never.

2. A client with left-sided heart failure is becoming increasingly short of breath. Which assessment finding is expected?
  1. Ascites
  2. Crackles
  3. Pedal pulses absent
  4. Cool fingers only

Break it down: Left side → blood backs into… lungs. Look for lung symptoms.

Show the answer

B — Crackles. Pressure backs up into the pulmonary veins, fluid leaks into alveoli, and you hear crackles in the bases first.

AAscites is fluid backing into the abdomen — that is right-sided failure.
CAbsent pedal pulses points to arterial occlusion or PAD, not heart failure.
DCool fingers alone is nonspecific and could be room temperature or vasoconstriction.

3. The monitor shows ventricular tachycardia. The client is unresponsive and has no pulse. Priority?
  1. Oxygen
  2. Defibrillation
  3. IV fluids
  4. ECG

Break it down: No pulse. ABC. Can blood circulate? No. Treat the rhythm immediately.

Show the answer

B — Defibrillation (with CPR until the defibrillator is ready). Pulseless VT is a shockable rhythm; only defibrillation stops the chaotic activity so a perfusing rhythm can return. Every minute of delay drops survival.

AOxygen is part of resuscitation but does nothing if no blood is circulating.
CFluids do not fix an electrical problem.
DYou already know the rhythm from the monitor. Ordering a 12-lead here only delays the shock.

4. A client with heart failure gains 5 lb (2.3 kg) in 3 days. What does this most likely indicate?
  1. Dehydration
  2. Fluid retention
  3. Improved nutrition
  4. Medication compliance

Break it down: Heart failure → poor pumping → fluid backs up. Weight gain = fluid until proven otherwise.

Show the answer

B — Fluid retention. 2.3 kg is roughly 2.3 L of retained fluid. Tissue cannot be gained that fast. This client needs the provider notified before she is in pulmonary edema.

ADehydration causes weight loss, not gain.
CReal tissue gain takes weeks, not 3 days.
DIf she were taking her diuretic correctly, weight would be stable or down.

5. Which client should the nurse assess FIRST?
  1. BP 168/94
  2. HR 108 after walking
  3. Chest pain rated 8/10 with diaphoresis
  4. Mild ankle swelling

Break it down: Ask: Who might be dying RIGHT NOW?

Show the answer

C — Chest pain 8/10 with diaphoresis. This is active myocardial ischemia. Muscle is dying while you decide.

AElevated but not a crisis (>180/120) and no organ-damage symptoms.
BAn expected response to activity in a client who just walked.
DChronic and slow. Needs assessment, not first.

6. A client with atrial fibrillation is at greatest risk for:
  1. Pneumonia
  2. Stroke
  3. GI bleed
  4. Renal stones

Break it down: AFib → blood pools → clot forms → where could it travel?

Show the answer

B — Stroke. The atria quiver instead of contracting, so blood stagnates in the left atrial appendage and clots. That clot leaves the left heart and the first major branches go to the brain. This is why these clients get anticoagulated.

ANot caused by the rhythm.
CA risk of the anticoagulant we give, not of the AFib itself — and it is not the greatest risk.
DNo relationship to atrial fibrillation.

7. A client reports chest pain only when climbing stairs and it resolves after resting. This is most consistent with:
  1. Stable angina
  2. Myocarditis
  3. Cardiac tamponade
  4. Pulmonary edema

Break it down: Exercise → pain → rest → pain gone. Think oxygen demand > oxygen supply.

Show the answer

A — Stable angina. Predictable, brought on by exertion, relieved by rest or nitroglycerin. The narrowed artery can supply a resting heart but not a working one.

BMyocarditis pain is constant and comes with fever and fatigue, not an exertion pattern.
CTamponade is an emergency: muffled heart sounds, JVD, hypotension. It does not come and go with stairs.
DPulmonary edema presents as severe dyspnea and crackles, not a resolving chest pain.

8. Which assessment finding suggests decreased cardiac output?
  1. Warm skin
  2. Urine output 15 mL/hr
  3. Bounding pulses
  4. BP 138/82

Break it down: Poor pump → poor kidney perfusion → low urine output.

Show the answer

B — Urine output 15 mL/hr. Below 30 mL/hr means the kidneys are not being perfused, which means forward flow has dropped. Urine output is your cheapest perfusion monitor.

AWarm skin means peripheral perfusion is intact — a normal finding.
CBounding pulses suggest volume overload or high output, not low output. Low output gives you weak, thready pulses.
D138/82 is only mildly elevated and does not indicate low output.

9. Which intervention is most important for a client experiencing acute pulmonary edema?
  1. Lay the client flat
  2. High-Fowler's position
  3. Encourage oral fluids
  4. Ambulate the client

Break it down: Pulmonary edema → fluid in lungs → improve breathing FIRST.

Show the answer

B — High-Fowler's position (upright, legs dependent). Sitting up lets the diaphragm drop and pools blood in the legs, which lowers preload and takes fluid off the lungs immediately. It costs nothing and works in seconds — that is why it comes before drugs.

ALying flat pushes more fluid into the lungs and worsens the dyspnea. Never lay a client in pulmonary edema flat.
CMore fluid is the opposite of what this client needs.
DAmbulating a hypoxic client in acute distress increases oxygen demand and risks collapse.

10. The nurse is caring for four cardiac clients. Which client is the priority?
  1. BP 150/88 taking morning medications
  2. Heart failure client with new confusion and oxygen saturation of 86%
  3. Stable atrial fibrillation with HR 82
  4. Client awaiting discharge after hypertension education

Break it down: Don't look at diagnoses. Look at: Who is losing oxygen? Who has poor perfusion? Who is unstable?

Show the answer

B — New confusion with SpO₂ 86%. Two failures at once: oxygenation and cerebral perfusion. New confusion is a change, and change is what the NCLEX rewards you for catching.

AElevated but being treated; no symptoms of organ damage.
CThe word "stable" with a controlled rate tells you this one is perfusing.
DDischarge teaching is important but nobody is unstable.

The 5 Questions to Ask on EVERY Cardiac NCLEX Question โญ

Before choosing an answer, ask yourself:

  1. What is the heart problem? Blocked artery, weak pump, rhythm problem, or high pressure — name it out loud.
  2. Can the heart still deliver oxygenated blood to the body? Look at LOC, BP, skin, pulses, urine output.
  3. Which assessment finding shows the patient is becoming unstable? The one that is new or trending the wrong way — confusion, falling SpO₂, rising RR, dropping urine.
  4. What threatens life first? Airway, breathing, circulation, then perfusion — in that order.
  5. Which intervention treats the underlying problem instead of the symptom? Fix the cause of the poor oxygen or poor perfusion; do not document, reposition, or reassess your way out of an emergency.

Slow down and answer these five before you read the options. You are looking for patterns, not facts — that is the skill being tested.

โญ If You Remember Nothing Else

Stop memorizing. Follow the chain.