Pulmonic stenosis Β· Aortic stenosis Β· Coarctation of the aorta β the OBSTRUCTIVE group
NG-183CARDIO Β· PEDSADHD-friendly visual edition
Nothing is leaking here β something is BLOCKED. A valve or a stretch of the aorta is
too narrow, so the chamber behind it has to squeeze harder and gets thick (hypertrophy).
Most of these babies stay pink. The one unmissable fingerprint of coarctation is a
blood pressure gap between the arms and the legs β high & bounding above, low & weak below.
π§ Nothing leaks β it's BLOCKEDA narrowed valve or vessel. The chamber behind the block hypertrophies pushing against it.
π Usually pinkOBSTRUCTIVE, not cyanotic β unless the block is critical and duct-dependent in a newborn.
π¦΅π€ 4-limb BP = coarctationArms: high BP, bounding pulses.Legs: low BP, weak/absent pulses. Check both arms too.
π¨ Critical = duct-dependentA severe block in a newborn may need the ductus arteriosus kept open (prostaglandin E1) until repair.
π«
THE BASE HEART
STEP 0 Β· SAME PICTURE AS EVERY PAGE
Same drawing as the ASD/VSD/PDA and Tetralogy pages. Nothing about the chambers changes here β the ROAD narrows, not the wall between them.
π« The normal heart β the drawing all four pages share
right heart Β· Oβ-poorleft heart Β· Oβ-richL β R shuntR β L shuntobstruction (bow-tie)
π§ βBlue in on the left of the page, red out on the right.β Right heart (blue) is drawn on your left,
left heart (red) on your right β because it is an anatomic view, facing the patient.
β THE classification you are tested on β this page lives in the OBSTRUCTIVE row
Group
Shunt direction
Pulmonary blood flow
Blue?
Defects
π ACYANOTIC
LEFT β RIGHT
β INCREASED β too much blood to the lungs
No (pink at first)
ASD Β· VSD Β· PDA Β· AVSD
π§‘ OBSTRUCTIVE β YOU ARE HERE
none β the road is narrowed
normal or β, depending on where the narrowing is
Usually no
Pulmonic stenosis Β· Aortic stenosis Β· Coarctation of the aorta
Transposition of the great arteries Β· Truncus arteriosus Β· TAPVR Β· hypoplastic left heart
π§ βPink holes, blue Ts, orange gates.β This page is the orange gates β
nothing is leaking through a hole, something is blocked, and the bow-tie βΆβ symbol marks exactly where.
π§ How to read every diagram in this series
Blue chambers = right heart, Oβ-poor blood.
Red chambers = left heart, Oβ-rich blood.
Citrine bow-tie βΆβ = an obstruction β the star of this page, three times.
Thick pale outline around a chamber = hypertrophy β that chamber is working against the block.
π§ Read the drawing right. It is an anatomic view, so the patient's
RIGHT heart is on YOUR LEFT. Same picture, four pages.
β Why an obstructive lesion causes trouble β one sentence
Answer first: pressure backs up behind the narrowing, and the chamber that has to push through it
gets thick and eventually tired.
Pulmonic stenosis backs pressure up into the right ventricle. Aortic stenosis backs pressure up into
the left ventricle. Coarctation backs pressure up into everything proximal to the pinch β the left
ventricle, and the vessels feeding the head and arms.
π§ βBehind the dam, the water rises.β Whatever sits before the narrowing takes the pressure hit.
π§
THE THREE BLOCKS
STEP 1 Β· SAME PICTURE, ONE PINCH EACH
Same base heart, three times. Only one thing changes each time β WHERE the road narrows.
What it is: the pulmonic valve (RV β pulmonary artery) is narrowed. The right ventricle
must generate much higher pressure to push blood into the lungs.
Classic sign: a systolic ejection murmur at the left upper sternal border, often with a
palpable thrill. Mild cases can be asymptomatic and found on a routine exam.
NCLEX TIPSeverity, not color, drives urgency. Mild/moderate pulmonic
stenosis stays pink and may just be watched. Critical pulmonic stenosis in a newborn can be
duct-dependent for pulmonary blood flow and needs prostaglandin E1 to keep the PDA open until
the valve is opened.
Fix:balloon valvuloplasty via cardiac catheterization is first-line for most children β
see NG-201 for cath care.
π§ βPinch the outflow, back up the room.β Pinch the pulmonary valve and the right ventricle
backs up pressure β and, over years, muscle.
2οΈβ£ AORTIC STENOSIS β narrowed aortic valve Β· OBSTRUCTIVE, left-sided, the higher-stakes one
What it is: the aortic valve (LV β aorta) is narrowed. The left ventricle β already the
strongest chamber β must work even harder to push blood out to the whole body.
Classic sign: a harsh systolic ejection murmur at the right upper sternal border /
aortic area, radiating to the neck/carotids. May have a weak, thready pulse and narrow pulse pressure
in severe cases.
NCLEX TRAP Because the LV is a systemic pump, severe aortic stenosis in a
newborn is an emergency β it can present as duct-dependent systemic circulation, with poor
perfusion, weak pulses, and shock-like presentation as the PDA closes.
Never assume an obstructive lesion is automatically low-priority β check the severity.
Fix:balloon valvuloplasty via catheterization, or surgical valve repair/replacement for
severe or recurrent disease.
π§ βThe strong pump meets a locked door.β Aortic stenosis makes the body's main pump fight
the hardest β that is why severe cases decompensate fastest.
3οΈβ£ COARCTATION OF THE AORTA β a pinch in the aorta itself Β· OBSTRUCTIVE β makes its own BP pattern
What it is: the aorta itself is narrowed, classically near where the ductus arteriosus
attaches β just past the arch, after the vessels to the head and arms have already branched off.
Classic signs: higher BP and bounding pulses in the arms, lower BP and weak or
absent pulses in the legs. Cool lower extremities. Older children may report leg cramps/fatigue
with exercise, headaches, or epistaxis; infants may present in shock when the PDA closes.
NCLEX TIPCheck BOTH arms, not just one. If the narrowing sits at or near
the origin of the left subclavian artery, the left arm BP can be lower than the right β a
right-arm-only BP can miss the picture.
Fix:balloon angioplasty Β± stent via catheterization, or surgical resection of the
narrowed segment. Lifelong follow-up β recoarctation and aneurysm risk persist into adulthood.
Associations: more common in Turner syndrome and with a bicuspid aortic valve β
both are worth knowing for exam questions.
π§ βGarden hose with a kink.β Everything before the kink sprays hard (arms β high
pressure); everything after the kink trickles (legs β low pressure).
One diagram, one idea: compare the arms to the legs. It is the single fastest way to catch coarctation at the bedside.
π¨ The hallmark assessment β BP and pulses in all four limbs
π§ βHigh up top, low down below.β Bounding radial pulses + high arm BP, paired with weak
or absent femoral pulses + low leg BP, is coarctation until proven otherwise.
β Radial-femoral delay β the pulse-timing trick
Answer first: palpate a radial pulse (wrist) and a femoral pulse (groin) at the same
time. Normally they arrive together.
In coarctation, the femoral pulse arrives noticeably later and weaker than the radial pulse β blood
is taking a slower, harder route around/through the narrowed segment to reach the legs.
π§ βThe femoral pulse is running late.β A delayed, weak femoral pulse next to a strong,
on-time radial pulse is a classic bedside finding β no equipment needed.
π Pulmonic vs Aortic vs Coarctation β tell them apart
Pulmonic stenosis
Aortic stenosis
Coarctation
Blocked chamber
Right ventricle
Left ventricle
Aorta itself
Murmur location
Left upper sternal border
Right upper sternal border, radiates to neck
May be absent or heard back/interscapular
Key exam clue
Murmur Β± thrill
Weak pulses, narrow pulse pressure if severe
Arm BP > leg BP, radial-femoral delay
Critical newborn risk
Duct-dependent pulmonary flow
Duct-dependent systemic flow β shock risk
Shock as PDA closes
π§ βP is for pulmonary (right), A is for aorta (left), C is for compare-the-limbs.β
β οΈ Why a βpinkβ diagnosis can still be an emergency
Obstructive lesions are usually acyanotic β but severe or critical narrowing in a newborn can be
duct-dependent: the baby only survives because the PDA is still open, letting blood bypass the
block.
As the ductus closes naturally in the first days of life, a critical obstruction can suddenly cause
shock, poor perfusion, and cardiovascular collapse β this is why every newborn gets pulses and perfusion
checked before discharge.
π§ βNo cyanosis β no emergency.β Watch perfusion and pulses, not just skin color, in a
newborn with a suspected obstructive lesion.
Two roads to relieve the block: catheter-based or surgical. Nursing priorities are the same for all three defects.
β How each block gets fixed
Defect
Catheter-based option
Surgical option
Pulmonic stenosis
Balloon valvuloplasty β usually first-line
Valve repair/replacement if balloon fails or valve is dysplastic
Aortic stenosis
Balloon valvuloplasty
Valve repair/replacement for severe or recurrent disease
Coarctation
Balloon angioplasty Β± stent
Surgical resection of the narrowed segment, especially in infants
SEE NG-201 for full pre- and post-catheterization care (pulse checks, straight-leg
positioning, site care) and post-surgical / chest tube care that applies after any of these repairs.
π§ βBalloon first, blade if it doesn't hold.β Catheter-based repair is usually tried
before open surgery for isolated valve or vessel narrowing.
π Watch for signs of heart failure β tachypnea, diaphoresis with feeds, poor weight gain β especially with moderate-severe stenosis.
3
π¨ Report poor perfusion in a newborn β weak/absent pulses, mottling, cool extremities, irritability or lethargy can mean the duct is closing on a critical lesion.
4
π Anticipate prostaglandin E1 orders for a duct-dependent critical lesion β keeps the PDA open until repair. (no dose stated here β follow facility protocol)
5
π Teach lifelong follow-up β recoarctation, aortic aneurysm, and hypertension can develop years later even after a successful repair.
π§ͺ What you'll see charted
Echocardiogram β confirms the diagnosis and severity (gradient across the valve/narrowing).
4-extremity BP documented as its own vital sign set when coarctation is suspected.
Pulse oximetry pre- and post-ductal (right hand vs a foot) can also help detect duct-dependent lesions.
π§ βPre vs post-ductal satsβ β right hand = pre-ductal, either foot = post-ductal. A gap between them points to a duct-dependent problem.
β Family teaching after repair
Keep all follow-up cardiology visits β narrowing can recur (recoarctation) as the child grows.
Endocarditis prophylaxis may be needed for certain dental/surgical procedures β ask the cardiologist which category applies.
Report headaches, nosebleeds, leg cramps with exercise, or a new BP gap between visits.
π§ βFixed once doesn't mean fixed forever.β A repaired coarctation still needs a cardiologist for life.
β‘
QUICK RECALL
SAY IT OUT LOUD
π§ Blocked, not leakingPulmonic, aortic, coarctation β all OBSTRUCTIVE. The chamber behind the block hypertrophies.
π¦΅π€ Arms high, legs lowCoarctation's fingerprint: high BP + bounding pulses (arms) vs low BP + weak/absent pulses (legs).
β±οΈ Radial-femoral delayFemoral pulse arrives late and weak compared to the radial pulse in coarctation.
π¨ Critical = duct-dependentWatch perfusion, not just color β a closing PDA can unmask a severe block fast in a newborn.
β One last time β the classification you will be asked
Group
Shunt direction
Pulmonary blood flow
Blue?
Defects
π ACYANOTIC
LEFT β RIGHT
β INCREASED β too much blood to the lungs
No (pink at first)
ASD Β· VSD Β· PDA Β· AVSD
π§‘ OBSTRUCTIVE β YOU ARE HERE
none β the road is narrowed
normal or β, depending on where the narrowing is
Usually no
Pulmonic stenosis Β· Aortic stenosis Β· Coarctation of the aorta