🀰 NUR 234 · Module 14

The High-Risk Newborn & Course Review

Exam 4 β€” Postpartum and the newbornWeek 14
πŸ“š Reading: ch. 21–26 β€” week split inferred
πŸ’‘ The one idea

Preterm complications are all about organs that had not finished. Lungs without surfactant, a gut without blood flow, eyes still forming vessels.

ComplicationBecauseWatch for
RDSNo surfactant before ~34–36 wksGrunting, nasal flaring, retractions, tachypnoea
NECGut ischemiaDistended abdomen, bloody stool, feeding intolerance
RetinopathyImmature retinal vessels + oxygenCareful O&sub2 titration
IVHFragile brain vesselsBulging fontanelle, apnea, ↓ tone
Feeding the newborn
🖼️ Feeding the newborn. Swipe it sideways if it is cut off, or tap to open it full size.

In suspected NEC, stop all feeds immediately and report. Feeding a hypoxic gut makes it worse.

Antenatal steroids given to a mother in preterm labor accelerate surfactant production β€” that single intervention is why RDS is far less lethal than it was.

πŸ’‰ The infant of a diabetic mother

Large baby, low sugar. Maternal glucose crossed the placenta; the baby’s pancreas ramped up insulin. At birth the sugar supply stops but the high insulin does not.

Check the glucose within the first hour and feed early. Also watch for birth injury from size, hypocalcemia and polycythemia.

🏁 Course-wide priorities to revise from
SituationFirst action
Membranes ruptureCheck the fetal heart rate
Late decelerationsTurn to the left side
Cord prolapseLift the presenting part off the cord
Boggy uterusMassage the fundus
Uterus displaced rightEmpty the bladder
Newborn just deliveredDry and warm
Preeclampsia + epigastric painNotify β€” seizure is imminent
Magnesium runningCheck deep tendon reflexes
Suspected previaNo vaginal exam

Cover the right column and say it out loud. If you can produce all nine, you have the spine of maternity nursing.

⭐ High-yield β€” what the exam actually asks

Show 5 moreHide these 5
  • RDS from surfactant deficiency: nasal flaring, grunting, retractions, tachypnea β†’ surfactant and respiratory support.
  • Meconium aspiration: tachypnea, retractions, grunting, cyanosis.
  • Prevent jaundice with early frequent feeding; phototherapy for a rising bilirubin.
  • SIDS risk reduction: back to sleep, no smoke exposure, firm flat surface, no co-sleeping.
  • Gestational diabetes: carbohydrate-controlled diet, exercise, frequent monitoring, glyburide/metformin or insulin. Insulin requirements climb through pregnancy as hPL rises, then fall abruptly after delivery. Fetal risks: macrosomia, shoulder dystocia, neonatal hypoglycemia.
Show 2 moreHide these 2
  • Cardiac disease in pregnancy: left lateral recumbent rest. The highest-risk window is immediately postpartum, as fluid shifts back. Report dyspnea, orthopnea, persistent cough, crackles, edema, fatigue.
  • The newborn is at hypothermia risk after a cesarean because the OR is cold β€” dry, cap, skin-to-skin, radiant warmer.

πŸ“• From your ATI review book

Covered by ch. 27.

Show 5 moreHide these 5
  • Classification: SGA below the 10th percentile Β· AGA 10th–90th Β· LGA above the 90th or over 4,000 g Β· low birth weight ≀2,500 g. Late preterm is 34 0/7–36 6/7.
  • NAS care is the opposite of stimulation: cluster care, dim lights, low noise, swaddle, non-nutritive sucking, small frequent high-calorie feeds, head elevated during and after feeds, suction available. Morphine, methadone, phenobarbital, sometimes clonidine.
  • NAS signs: high-pitched shrill cry, tremors, hypertonicity, exaggerated Moro, frantic uncoordinated sucking with poor intake, diarrhea, projectile vomiting, sneezing, yawning, mottling.
  • Fetal alcohol syndrome: small palpebral fissures, flat midface, smooth philtrum, thin upper lip, plus growth restriction. No safe amount.
  • RDS: tachypnea over 60, nasal flaring, expiratory grunting, retractions, fine crackles, cyanosis. Worsening looks like flaccidity, apnea and unresponsiveness. Birth weight alone does not predict lung maturity.
Show 5 moreHide these 5
  • After surfactant, do NOT suction the ET tube for 1–4 hours. Suction before, not after.
  • Before any nipple or breast feeding the newborn needs an intact gag and a coordinated suck-swallow; under 34 weeks generally means IV or gavage.
  • Shoulder dystocia: McRoberts position and SUPRAPUBIC pressure β€” never fundal pressure.
  • Erb-Duchenne palsy: flaccid arm, extended elbow, inward-rotated hand, absent Moro on that side, grasp reflex INTACT. Clavicle fracture: limited motion, crepitus, absent Moro.
  • Postmature: wasted look, loose peeling leathery skin, long nails and hair, meconium staining, alert like a 2-week-old, hypoglycemia from depleted glycogen. Highest risks are meconium aspiration and persistent pulmonary hypertension.
Show 5 moreHide these 5
  • Jaundice is a TIMING question: after 24 h = physiologic. Before 24 h = pathologic, usually blood-group incompatibility or infection. ABO incompatibility = parent type O, newborn A or B; a positive direct Coombs confirms.
  • Phototherapy: eye mask on, undressed except for a diaper, reposition every 2–3 h so every surface gets treated, unmask q4h to check for injury, temp q4h. Turn the lights OFF before drawing bilirubin. Bronze discoloration and a maculopapular rash are expected β€” not a reason to stop. No lotion or oil on the skin (burns under the lights) and no water or glucose water β€” it displaces milk, so fewer stools and a higher bilirubin. Feed 8–12 times a day to move bilirubin out in the stool; levels should fall in 4–6 h.
  • Kernicterus is irreversible. Acute bilirubin encephalopathy β€” lethargy, hypotonia, arching, high-pitched cry β€” is the reversible stage before it.
  • Neonatal sepsis is subtle: temperature INSTABILITY rather than fever, poor feeding, lethargy, glucose swings, apnea, color change, hypotonia.
  • TEF/esophageal atresia: excessive oral secretions, drooling, choking and cyanosis with feeding, often preceded by polyhydramnios. Withhold feedings, head up, orogastric tube to low continuous suction.
Show 1 moreHide these 1
  • Positioning by anomaly: myelomeningocele prone with a sterile moist dressing Β· tetralogy knee-chest during a spell Β· cleft lip/palate upright with frequent burping.

📚 From your Maternal & Child textbook

Pillitteri, Maternal and Child Health Nursing — ch. 26 (high-risk newborn) · ch. 27 (physical or developmental difference).

  • Corticosteroids in preterm labor are for the fetal lungs, not for the labor. They accelerate surfactant formation and reduce respiratory distress syndrome.

⚠️ Exam traps

  • For a cardiac client the dangerous part is after delivery, not labor.
  • Postpartum insulin needs drop off a cliff. Yesterday's correct dose causes hypoglycemia today.

🧠 Mind maps 1

One per disorder, built from the structure of your ATI chapter.

Complications of the Newborn
🎯 Who gets it
  • Maternal diabetes raises risk for newborn hypoglycemia, RDS, and being large for gestational age.
  • Preterm birth risk factors: gestational hypertension, PPROM, cervical insufficiency, placenta previa, and a prior preterm delivery.
  • SGA risk factors: maternal malnutrition or infection, smoking or substance use, placental insufficiency, or fetal congenital infection like rubella.
  • Macrosomic (LGA) risk factors: postmaturity, maternal obesity, genetics, and poorly controlled maternal hyperglycemia.
πŸ‘€ What you see
  • RDS: tachypnea over 60/min, grunting, retractions, nasal flaring, and cyanosis.
  • Preterm: Ballard score under 37 weeks, heavy lanugo, weak or absent suck/swallow/gag, apnea lasting 20 sec or more.
  • SGA: weight under the 10th percentile, normal head size but a thin body and little subcutaneous fat.
  • LGA: weight over the 90th percentile (past 4,000 g), birth trauma, and tremors from low calcium.
πŸ§ͺ What confirms it
  • NAS is confirmed by testing urine, meconium, hair, or umbilical cord tissue for drug exposure.
  • Confirm hypoglycemia with a lab glucose sample after a bedside reading under 45 mg/dL.
  • RDS labs: ABGs, CBC with differential, and blood or CSF cultures.
  • Preterm labs often show low Hgb/Hct and prolonged PT/aPTT, raising bleeding risk.
🩺 What you do
  • Feed a stable, hypoglycemic newborn within an hour of birth, then recheck glucose and feed roughly every 2 to 3 hr across the first 24 hr.
  • Confirm an intact gag, suck, and swallow reflex before oral feeding a preterm newborn to avoid aspiration.
  • Use a radiant warmer or isolette and cluster care to limit handling in preterm, SGA, or postmature newborns.
  • Encourage kangaroo (skin-to-skin) care to lower preterm infant stress and support temperature control.
πŸ’Š Drugs
  • Morphine sulfate, a short-acting opioid analgesic, treats neonatal withdrawal symptoms.
  • Methadone, a long-acting synthetic opioid, is used for neonatal opioid withdrawal.
  • Phenobarbital (anticonvulsant) lowers CNS irritability and controls seizures from alcohol or opioid withdrawal; clonidine may be added.
  • Surfactant replacement (beractant, calfactant, or lucinactant) restores lung surfactant in preterm RDS.
πŸ’¬ What you teach
  • Encourage NAS-affected families to enter drug/alcohol treatment and follow SUID-prevention practices.
  • Encourage preterm and SGA parents to take an active role in care and plan ahead for home needs.
  • Hyperbilirubinemia follow-up: low-to-moderate risk newborns within 2 days, higher-risk newborns within 24 hr of discharge.
⚠️ What goes wrong
  • Preterm RDS involves reduced alveolar surfactant regardless of the newborn's birth weight.
  • Bronchopulmonary dysplasia: ventilator/oxygen therapy overinflates and damages immature lungs over time.
  • Apnea of prematurity comes from immature neurologic and chemical breathing control.
  • Intraventricular hemorrhage is bleeding in or around the brain's ventricles.

Read left to right: who gets it β†’ what you see β†’ what confirms it β†’ what you do β†’ what goes wrong. Cover a column and rebuild it out loud.

🎥 Lecture recordings 4

Tap a card to open that recording in Google Drive. The same list lives in the lecture library.

All NUR 234 recordings →

πŸ–ΌοΈ Infographics 16

Tap a card to open the matching graphics in your infographic library.

A newborn outline with four markers on it β€” IVH at the brain, RDS at the lungs, NEC at the gut and cold stress at the skin β€” and a panel for each explaining the signs and the nursing care.
Four systems fail at once in prematurity. The marker shows you where each one is, and the panel beside it gives the signs and what you do. — tap it to open full size.
πŸ–ΌοΈHyperbilirubinemia & HypothermiaOpen matching infographics β†’πŸ–ΌοΈHypoxia & HypoglycemiaOpen matching infographics β†’
The glucose threshold of 40 mg per deciliter on a scale, who is at risk of newborn hypoglycemia, the six non-specific signs and what you do first.
Jittery plus a mother with diabetes equals a glucose check. Feed her first; IV dextrose only if she cannot suck or stays low. — tap it to open full size.
A newborn drawn with jaundice spreading head to toe and the approximate bilirubin level at each landmark, a table separating physiologic from pathologic jaundice, and the phototherapy rules.
Yellow in the first 24 hours is never normal. After that, when it started is what tells you which kind it is. — tap it to open full size.
πŸ–ΌοΈSIDS & Neonatal SepsisOpen matching infographics β†’📚Toxoplasmosi & TORCH InfectionsOpen the full study page →

📄 Simple Nursing handouts for this module — 9 of them, straight from your Drive.

🖼️ See all 67 NUR 234 handouts in the visual library →

πŸ“‹ Active Learning Templates 1

One per disorder. Every row is filled from that section of the ATI chapter β€” print it, cover the right, rebuild it.

📋 Complications of the Newborn6 parts

🖼️ InfographicsThe High-Risk NewbornToxoplasmosi & TORCH InfectionsPreterm Labor InterventionsDIC & Meconium AspirationHyperbilirubinemia & HypothermiaHypoxia & HypoglycemiaNRP & NECFTT & FASNAS & RDSSIDS & Neonatal Sepsis

ATI Active Learning Template β€” System DisorderComplications of the Newborn

Filled from ATI chapter 27, row by row from that chapter’s own sections β€” 12 of 12 rows have content.

1 rows came from outside your ATI chapter β€” 1 cite a source, 0 are built from this page’s own notes. Each one is labeled.

🧭 What it isAlterations in Health (Diagnosis) · Health Promotion & Disease Prevention
Alterations in Health (Diagnosis)
  • Surveys major newborn complications: substance withdrawal, hypoglycemia, respiratory distress, prematurity, growth extremes, tracheesophageal fistula, sepsis, birth trauma, hyperbilirubinemia, and congenital anomalies, covering risk factors, expected findings, diagnostics, and nursing priorities for early recognition.
Health Promotion & Disease Prevention
  • TEF can often be identified on a prenatal ultrasound before birth.
πŸ‘€ How it shows upAssessment β€” Risk Factors Β· Assessment β€” Expected Findings
Assessment β€” Risk Factors
  • Maternal diabetes raises risk for newborn hypoglycemia, RDS, and being large for gestational age.
  • Preterm birth risk factors: gestational hypertension, PPROM, cervical insufficiency, placenta previa, and a prior preterm delivery.
  • SGA risk factors: maternal malnutrition or infection, smoking or substance use, placental insufficiency, or fetal congenital infection like rubella.
  • Macrosomic (LGA) risk factors: postmaturity, maternal obesity, genetics, and poorly controlled maternal hyperglycemia.
  • A pregnancy past 40 weeks is usually of unknown cause but occurs more with first pregnancies or a prior postmature birth.
  • TEF risk factors: prior polyhydramnios, cardiac anomaly, cleft lip/palate, or a neural tube defect.
  • Newborn sepsis risk: ruptured membranes, prolonged labor, TORCH infection, prematurity, low birth weight, or invasive lines.
  • Birth trauma risk: fetal macrosomia, difficult presentation, precipitous or prolonged labor, CPD, or forceps/vacuum delivery.
Assessment β€” Expected Findings
  • RDS: tachypnea over 60/min, grunting, retractions, nasal flaring, and cyanosis.
  • Preterm: Ballard score under 37 weeks, heavy lanugo, weak or absent suck/swallow/gag, apnea lasting 20 sec or more.
  • SGA: weight under the 10th percentile, normal head size but a thin body and little subcutaneous fat.
  • LGA: weight over the 90th percentile (past 4,000 g), birth trauma, and tremors from low calcium.
  • Postmature: wasted look, cracked/leathery/peeling skin, long nails, alertness resembling a newborn already 2 weeks old.
  • NAS: shrill cry, tremors, increased muscle tone, poor feeding, and uncoordinated sucking.
  • Sepsis: unstable temperature, poor feeding, apnea, skin color changes, irritability, and low muscle tone.
  • Birth injury clues: a hoarse cry suggests laryngeal nerve palsy; an absent Moro reflex with an inward-turned arm suggests Erb-Duchenne palsy.
πŸ§ͺ How it is confirmedLaboratory Tests Β· Diagnostic Procedures
Laboratory Tests
  • NAS is confirmed by testing urine, meconium, hair, or umbilical cord tissue for drug exposure.
  • Confirm hypoglycemia with a lab glucose sample after a bedside reading under 45 mg/dL.
  • RDS labs: ABGs, CBC with differential, and blood or CSF cultures.
  • Preterm labs often show low Hgb/Hct and prolonged PT/aPTT, raising bleeding risk.
  • LGA newborns often show polycythemia on CBC, with Hct above 65%, from chronic hypoxia.
Diagnostic Procedures
  • Chest x-ray screens for RDS, meconium aspiration, and (in FAS) congenital heart defects.
  • Preterm newborns get head ultrasound, echocardiography, and eye exams to catch complications early.
  • TEF may show on prenatal ultrasound; confirm after birth with a chest x-ray and NG tube study.
  • Birth injuries are diagnosed with CT, targeted x-ray, or a neurologic exam.
  • Transcutaneous bilirubin testing estimates bilirubin level without a blood draw.
🩺 What you doNursing Care · Medications · Therapeutic Procedures
Nursing Care
  • Feed a stable, hypoglycemic newborn within an hour of birth, then recheck glucose and feed roughly every 2 to 3 hr across the first 24 hr.
  • Confirm an intact gag, suck, and swallow reflex before oral feeding a preterm newborn to avoid aspiration.
  • Use a radiant warmer or isolette and cluster care to limit handling in preterm, SGA, or postmature newborns.
  • Encourage kangaroo (skin-to-skin) care to lower preterm infant stress and support temperature control.
  • TEF: keep the head of bed raised, maintain low continuous orogastric suction, and withhold feeds until a provider evaluates secretions or distress.
  • Avoid endotracheal suctioning for 1 to 4 hr after giving surfactant.
  • During phototherapy, shield the eyes, remove the mask every 4 hr to inspect them, change the newborn's position every 2 to 3 hr, and turn off lights before blood draws. No lotions or oils on the skin, and no supplemental water or glucose water.
  • During phototherapy, feed early and often, about 8 to 12 times a day, to help clear bilirubin through the stool.
  • Give an exchange transfusion for newborns at risk of kernicterus from severe hyperbilirubinemia.
Medications
  • Morphine sulfate, a short-acting opioid analgesic, treats neonatal withdrawal symptoms.
  • Methadone, a long-acting synthetic opioid, is used for neonatal opioid withdrawal.
  • Phenobarbital (anticonvulsant) lowers CNS irritability and controls seizures from alcohol or opioid withdrawal; clonidine may be added.
  • Surfactant replacement (beractant, calfactant, or lucinactant) restores lung surfactant in preterm RDS.
  • Antireflux drugs and antacids help manage TEF-related reflux.
  • Thyroid hormone replacement treats congenital hypothyroidism.
Therapeutic Procedures
  • Surgical repair corrects the specific TEF/EA defect.
  • Phototherapy should begin lowering bilirubin within 4 to 6 hr of starting.
πŸ’¬ Around the patientClient Education Β· Interprofessional Care
Client Education
  • Encourage NAS-affected families to enter drug/alcohol treatment and follow SUID-prevention practices.
  • Encourage preterm and SGA parents to take an active role in care and plan ahead for home needs.
  • Hyperbilirubinemia follow-up: low-to-moderate risk newborns within 2 days, higher-risk newborns within 24 hr of discharge.
  • PKU is an inability to break down phenylalanine; galactosemia is an inability to convert galactose to glucose.
  • Down syndrome is trisomy 21, the most common trisomy, with 47 chromosomes in each cell.
  • Tetralogy of Fallot combines a VSD, an overriding aorta, pulmonary stenosis, and right ventricular hypertrophy.
  • Patent ductus arteriosus is a noncyanotic defect in which the ductus stays open instead of closing after birth.
Interprofessional Care

Not in your ATI chapter β€” filled from StatPearls, 2026.

  • Physicians and advanced practice providers adjust antibiotic choices based on culture results and how the infant is responding.
  • Bedside nurses handle continuous monitoring for early warning signs and keep the family informed and involved in care.
  • Pharmacists help select and dose antimicrobials correctly, supporting safe and appropriate antibiotic use.
  • Social workers and lactation consultants get pulled in too, supporting the family and planning ahead for discharge.

StatPearls (NCBI Bookshelf) Β· Neonatal Sepsis Β· open the source β†’

⚠️ What goes wrongComplications
Complications
  • Preterm RDS involves reduced alveolar surfactant regardless of the newborn's birth weight.
  • Bronchopulmonary dysplasia: ventilator/oxygen therapy overinflates and damages immature lungs over time.
  • Apnea of prematurity comes from immature neurologic and chemical breathing control.
  • Intraventricular hemorrhage is bleeding in or around the brain's ventricles.
  • Retinopathy of prematurity involves abnormal retinal vessel growth and can impair vision.
  • Patent ductus arteriosus is a noncyanotic defect where the ductus stays open after birth.
  • Necrotizing enterocolitis is ischemic bowel injury that can progress to necrosis or perforation.

πŸ“ Notes & key concepts

The lines from this module that carry a number, a dose or an absolute rule β€” the ones that decide questions. Everything else is on the cards above.

  • Classification: SGA below the 10th percentile Β· AGA 10th–90th Β· LGA above the 90th or over 4,000 g Β· low birth weight ≀2,500 g. Late preterm is 34 0/7–36 6/7.
  • RDS: tachypnea over 60, nasal flaring, expiratory grunting, retractions, fine crackles, cyanosis. Worsening looks like flaccidity, apnea and unresponsiveness. Birth weight alone does not predict lung maturity.
  • After surfactant, do NOT suction the ET tube for 1–4 hours. Suction before, not after.
  • Before any nipple or breast feeding the newborn needs an intact gag and a coordinated suck-swallow; under 34 weeks generally means IV or gavage.
  • Shoulder dystocia: McRoberts position and SUPRAPUBIC pressure β€” never fundal pressure.
  • Postmature: wasted look, loose peeling leathery skin, long nails and hair, meconium staining, alert like a 2-week-old, hypoglycemia from depleted glycogen. Highest risks are meconium aspiration and persistent pulmonary hypertension.
  • Jaundice is a TIMING question: after 24 h = physiologic. Before 24 h = pathologic, usually blood-group incompatibility or infection. ABO incompatibility = parent type O, newborn A or B; a positive direct Coombs confirms.
  • Phototherapy: eye mask on, undressed except for a diaper, reposition every 2–3 h so every surface gets treated, unmask q4h to check for injury, temp q4h. Turn the lights OFF before drawing bilirubin. Bronze discoloration and a maculopapular rash are expected β€” not a reason to stop. No lotion or oil on the skin (burns under the lights) and no water or glucose water β€” it displaces milk, so fewer stools and a higher bilirubin. Feed 8–12 times a day to move bilirubin out in the stool; levels should fall in 4–6 h.

🎯 Module quiz

Questions for this module. They also feed the Mega Quiz.

Nothing here yet β€” drop it in when you have it