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Nursing Field Notes / Integumentary Β· Skin Infections & Inflammatory Conditions Β· Pathophysiology Course

Impetigo 🍯

The honey-crusted rash β€” highly contagious, mostly in children

NG-209 INTEGUMENTARY Β· PEDS INFECTION ADHD-friendly visual edition

Impetigo is a very common, highly contagious superficial bacterial skin infection of infants and children, caused by Staph aureus and/or Group A Strep. Itchy red sores around the nose and mouth burst and dry into the unmistakable honey-colored crust β€” and that crusting stage is when it spreads fastest, by hands, towels, linen and toys. Nursing care is half skin care and half stopping the spread, plus one safety-net: strep in the skin can later reach the kidneys.

📄 Simple Nursing original — opens in Drive →

🍯 Honey-colored crustThe single most recognizable sign in dermatology. Golden, stuck-on crust around the nose and mouth of a child.
🚨 HIGHLY contagiousDirect contact and objects β€” towels, linen, toys, clothes. The crusting stage is the most contagious.
🦠 Staph or StrepSuperficial β€” top of the epidermis only, so it heals without scarring (except the deep form, ecthyma).
🏫 Home from schoolOff school/daycare, hand hygiene, separate laundry in hot water, short nails, gently soak crusts off.
🧬

THE SKIN MODEL

STEP 0 Β· LEARN THIS ONCE

Same skin drawing as every page in this set β€” this time with a shallow blister forming right under the surface.

πŸ”¬ Skin cutaway β€” impetigo is the SHALLOW one

DEPTH = THE ANSWER The blister forms under the stratum corneum, so its roof is paper-thin. It ruptures almost immediately, and the fluid dries into the honey-colored crust. Because it never reaches the dermis, ordinary impetigo does not scar.

IMPETIGO Β· the same skin model, infected Staph or Strep multiply just under the stratum corneum β€” a shallow blister that bursts and dries into crust. Stratum corneum dead keratin β€” the barrier Granular + spinous layers living keratinocytes Basal layer & rete ridges new cells are born here Papillary dermis capillary loops Reticular dermis collagen, glands, follicles Subcutaneous fat adipose lobules Fascia / muscle hair shaft sebaceous gland hair bulb sweat gland coil arteriole venule touch receptor sensory nerve EPIDERMIS ONLY β‘  thin-walled vesicle just UNDER the stratum corneum β‘‘ it bursts β†’ HONEY CRUST the MOST contagious moment onto hands, towels, toys, bed linen β†’ IMPETIGO stays SHALLOW: top of the epidermis only Shallow = heals WITHOUT scarring. Deep (ecthyma) = it scars.
blister fluid / pus honey crust staph & strep cocci neutrophils
🧠 "Impetigo is a roof problem." The infection is in the roof of the skin, so it peels and crusts instead of swelling and spreading like cellulitis.

πŸ“– Lesion vocabulary β€” impetigo uses four of these words

Impetigo is a vesicle β†’ pustule β†’ crust story, and the bullous form adds the bulla. Highlighted below.

LESION MORPHOLOGY β€” the vocabulary behind every skin question Side-on cutaway (top) + what you see looking down at the skin (bottom). MACULE flat, color only Β· < 1 cm SIDE VIEW β–Ύ LOOKING DOWN β–Ύ e.g. freckle PAPULE solid raised bump Β· < 1 cm SIDE VIEW β–Ύ LOOKING DOWN β–Ύ e.g. insect bite PLAQUE raised flat-topped Β· > 1 cm SIDE VIEW β–Ύ LOOKING DOWN β–Ύ e.g. psoriasis VESICLE clear fluid blister Β· < 1 cm SIDE VIEW β–Ύ LOOKING DOWN β–Ύ e.g. shingles BULLA big fluid blister Β· > 1 cm SIDE VIEW β–Ύ LOOKING DOWN β–Ύ e.g. bullous impetigo PUSTULE pus-filled Β· any size SIDE VIEW β–Ύ LOOKING DOWN β–Ύ e.g. impetigo CRUST dried exudate Β· on top SIDE VIEW β–Ύ LOOKING DOWN β–Ύ e.g. honey crust SCALE flaking keratin Β· on top SIDE VIEW β–Ύ LOOKING DOWN β–Ύ e.g. tinea
🧠 Vesicle is small, bulla is big. "B" for BIG bulla.
🦠

PATHO & CAUSES

STEP 1 Β· WHY IT HAPPENS

Common bacteria + a child's skin + a scratch = the most contagious rash on the ward.

🧠 The one-sentence definition

Impetigo is a very common, highly contagious superficial bacterial skin infection caused by Staphylococcus aureus and/or Group A Streptococcus, affecting mostly infants and young children.

It lives in the top of the epidermis β€” that is why it crusts rather than swells, and why it usually heals without a scar.

🧠 "Impetigo β€” infant-igo." The name itself tells you the age group.

βš™οΈ Mechanism, step by step

Bacteria colonize the nose & skin surface
β–Ό
A tiny break β€” scratch, bite, eczema, cut
β–Ό
Bacteria multiply just under the stratum corneum
β–Ό
A thin-walled vesicle fills with pus
β–Ό
It ruptures β†’ oozes β†’ dries into HONEY-COLORED CRUST
β–Ό
The child scratches β†’ new lesions + other children
🧠 Itch is the engine. Scratching is what seeds the next lesion and the next child β€” which is why short nails is a real intervention, not a nicety.

⭐ Who gets it & what makes it worse

  • Age 2–5 years is the classic peak; can occur at any age
  • Crowding β€” daycare, school, siblings, sports teams
  • Warm, humid weather and summer months
  • Broken skin β€” eczema, insect bites, chickenpox, scabies, minor trauma
  • Poor hygiene or limited access to washing facilities
  • Skin-to-skin contact sports β€” wrestling, rugby
🧠 "Hot, crowded, scratched." Those three words cover nearly every risk factor.

🚨 How it travels β€” and the five places you break the chain

HOW IT TRAVELS β€” and the five places you can break the chain Direct skin contact AND contaminated objects (fomites). This is why impetigo sweeps through a daycare room. INFECTED CHILD scratches β†’ bacteria on fingers 🧺 Bed linen 🧻 Towels πŸ‘• Clothes 🧸 Toys 🀝 Skin-to-skin NEXT CHILD new lesions in 1–3 days
🧠 Fomite = an object that carries the bug. Towel, toy, doorknob, wrestling mat. Impetigo spreads by contact, not by air.

πŸ§ͺ Bug β†’ drug: the rule that runs this whole set

NAME THE ORGANISM β†’ THE DRUG CLASS NAMES ITSELF This one rule answers most skin-infection questions. The suffix tells you the class. β—€ THIS PAGE β–Ά BACTERIAL clusters = staph chains = strep Staph aureus Β· Strep pyogenes ANTIBIOTIC cephalexin Β· dicloxacillin Β· clindamycin Β· mupirocin (topical) Β· doxycycline or TMP-SMX if MRSA suspected Cellulitis Β· Impetigo FUNGAL branching hyphae + spores dermatophytes (Trichophyton) ANTIFUNGAL topical "-azole" (clotrimazole, miconazole, ketoconazole) Β· terbinafine Β· oral terbinafine / griseofulvin for scalp & nails Tinea (ringworm) VIRAL enveloped DNA virus HSV-1 / HSV-2 Β· varicella-zoster ANTIVIRAL the "-cyclovir" family: acyclovir Β· valacyclovir Β· famciclovir β€” start early, they shorten but do not cure Cold sores Β· Shingles AUTOIMMUNE T cell attacks keratinocytes the patient's own T cells IMMUNOMODULATION topical corticosteroids Β· vitamin D analogue (calcipotriene) Β· phototherapy Β· methotrexate Β· biologics (e.g. infliximab) Psoriasis Β· Eczema
🧠 Bacteria β†’ antibiotic. The only extra decision in impetigo is topical or oral β€” and that is decided by how much skin is involved.
πŸ”Ž

CLUES

STEP 2 Β· WHAT YOU SEE

A child, itchy sores around the nose and mouth, and a crust the color of dried honey.

🍯 The classic picture

THE CLASSIC PICTURE β€” a child, around the nose and mouth Itchy red sores that ooze and dry to a stuck-on, golden crust. Most common in infants and young children. HONEY-COLORED CRUST β€” up close Stuck-on, golden-yellow, "corn-flake" crust Underneath it is moist, red and weeping.
🧠 "Golden crust on a small face." If the stem says honey-colored or golden-yellow crust in a child, the answer is impetigo β€” every time.

πŸ‘€ Signs & symptoms

  • Itchy red sores, classically around the nose and mouth; also hands, feet, arms and legs
  • Sores burst and dry into a stuck-on, honey/golden crust
  • Lesions at different stages at the same time β€” new ones keep appearing
  • Regional lymph nodes may be swollen and tender
  • Usually little or no fever and the child is otherwise well
  • Itch, not pain β€” pain suggests the deeper form (ecthyma) or cellulitis
🧠 Itchy + crusty + a well child = impetigo. Painful + hot + an unwell child = think cellulitis.

πŸ§ͺ Diagnosis & work-up

  • Mostly clinical β€” the appearance is the diagnosis
  • Swab for culture & sensitivity if it is widespread, recurrent, not responding, or MRSA is suspected
  • Consider checking for an underlying cause: eczema, scabies, head lice, chickenpox
  • If PSGN is suspected later: urinalysis (blood + protein), BP, and renal function
🧠 Swab before starting the antibiotic if a swab is being taken at all.

πŸ“ˆ How one sore evolves β€” and its most contagious moment

HOW ONE SORE EVOLVES β€” and when it is most contagious Each lesion runs this course in a few days. New lesions keep appearing while old ones crust β€” so you see all stages at once. β‘  MACULE red flat spot the itch starts here scratching seeds more β‘‘ VESICLE thin-walled blister roof is paper-thin so it bursts fast β‘’ PUSTULE fills with pus neutrophils pile in fluid turns cloudy β‘£ RUPTURE bursts, oozes 🚨 MOST CONTAGIOUS the fluid is loaded with live bacteria β‘€ CRUST dries golden 🚨 STILL CONTAGIOUS crust still sheds bacteria β€” keep covered
🧠 The crust is not the "healed" stage. Students lose marks here: a crusted lesion is still shedding bacteria.

πŸ”¬ Three forms β€” non-bullous vs bullous vs ecthyma

THREE FORMS β€” the difference is DEPTH Shallow forms heal clean. The deep form leaves a scar β€” that is the whole reason to know them apart. NON-BULLOUS ~70% of cases Β· Staph or Strep epidermis dermis fat Small vesicles that rupture fast and leave the classic honey crust. Around the nose and mouth. Heals WITHOUT scarring. BULLOUS Staph aureus toxin epidermis dermis fat > 1 cm Large flaccid BULLAE (> 1 cm) with clear then cloudy fluid, on the trunk, arms, diaper area. Common under 2 years. Heals WITHOUT scarring. ECTHYMA the deep, ulcerating form epidermis dermis fat SCARS when it heals Punches through into the DERMIS: a painful, punched-out ulcer with a thick crust and a raised violet edge. DOES scar.
NON-BULLOUSBULLOUSECTHYMA
DepthSuperficial epidermisSuperficial epidermisInto the dermis
LesionSmall vesicles β†’ honey crustLarge flaccid bullae > 1 cmPunched-out ulcer, thick crust
WhereFace, around nose/mouthTrunk, arms, diaper areaLegs, buttocks
PainItchyItchy, may be tenderPainful
Scars?NoNoYes
TreatmentTopical, or oral if extensiveUsually oral antibioticOral antibiotic
🧠 "Ecthyma etches." Only the deep one leaves a mark.

🚨 The complication that matters β€” PSGN

THE COMPLICATION THAT MATTERS β€” strep skin infection β†’ the kidney Post-streptococcal glomerulonephritis (PSGN). It typically appears 1–3 weeks AFTER the skin infection, when the sores may already look better. β‘  SKIN INFECTION Group A Streptococcus in the skin lesion The body makes antibodies against the strep. Antibody + antigen stick together = immune complex β‘‘ TRAPPED IN THE GLOMERULUS afferent efferent immune complexes clog the filter β†’ blood + protein leak into urine Filtration falls β†’ fluid is retained β†’ puffiness and a rising blood pressure. β‘’ WHAT THE FAMILY WILL SEE TEA / COLA urine 🫧 Puffy face & eyelids πŸ“ˆ High blood pressure πŸ’§ Less urine than usual 😴 Tired, off food, fever βš–οΈ Sudden weight gain ⏱️ TIMING: ~1–3 weeks after the skin sores Antibiotics limit spread to other children but do not reliably prevent PSGN β€” so teach the family what to watch for after the rash heals. Rheumatic fever follows strep THROAT, not skin.
🧠 "Throat β†’ heart. Skin β†’ kidney." Strep throat can lead to rheumatic fever; strep skin infection is the one linked to glomerulonephritis.
🩺

CARE

STEP 3 Β· WHAT YOU DO

Treat the skin, and stop it reaching the rest of the class.

βœ… Stop the spread β€” the five teaching points

STOP THE SPREAD β€” the five teaching points, in order These are the highest-yield exam answers on this page. Say them in this order and you will not miss one. 1 🏫 NO school or daycare Stay home until 24–48 h of antibiotic treatment and until lesions are drying and healing β€” follow the local public-health rule. 2 🧼 Wash hands Before AND after touching the sores, applying ointment or changing dressings. Everyone in the house, every time. 3 🧺 Separate the laundry The child's clothes, towels and bed linen get washed separately, in HOT water, and are never shared. 4 πŸ’… Short, filed nails Cut them short and file them smooth. Scratching spreads bacteria to new skin and drives it deeper. 5 🧽 Soak off the crust Soften crusts with a warm, soapy soak, remove gently, then apply the prescribed ointment to clean skin. Cover loosely.
🧠 "School · Soap · Separate · Short nails · Soak." Five S's, in the order you would teach them.

πŸ’Š Topical or oral? The decision rule

TOPICAL β€” for a few localized lesions: mupirocin ointment applied to the lesions after gently removing crust. (Retapamulin and ozenoxacin are alternatives where available.)

ORAL β€” when it is widespread, bullous, ecthyma, involves multiple household members, or has failed topical therapy: a cephalosporin (cephalexin) or an anti-staph penicillin; clindamycin or TMP-SMX / doxycycline if MRSA is suspected.

🧠 "A few = cream. A lot = pill."

🧽 Crust care β€” the technique

  • Gloves on. Standard + contact precautions for draining lesions
  • Soak with warm soapy water or a saline compress to soften the crust
  • Gently lift the softened crust β€” do not scrub or pick at dry crust
  • Pat dry, then apply the ointment to clean skin β€” ointment on top of crust does not reach the bacteria
  • Cover loosely with a non-adherent dressing to stop scratching and shedding
  • Dispose of dressings as infectious waste; hand hygiene after removing gloves
🧠 Soften, lift, treat, cover. Ointment on crust is wasted ointment.

πŸ—£οΈ Family teaching β€” say it exactly like this

  • "It is very catching β€” mostly by touch and by shared things."
  • "Keep them home from school/daycare until the treatment has been in for 24–48 h and the sores are drying up."
  • "Wash your hands before and after touching the sores or putting on the cream."
  • "Their own towel, their own bedding β€” washed separately in hot water."
  • "Nails short and filed, and try to keep the sores covered."
  • "Finish the whole course, even when it looks better."
  • "Come back if the sores spread, get painful, or the child develops fever, puffy eyes, or dark, tea-colored urine."
🧠 The last bullet is the PSGN safety-net. Never end impetigo teaching without it.

❌ Never do these

  • Never let the child share towels, bedding, washcloths or clothing.
  • Never scrub or forcibly pick dry crusts β€” soften them first.
  • Never send the child back to school just because the sores look drier.
  • Never apply ointment on top of a thick crust and call it treated.
  • Never skip hand hygiene between lesion care and any other patient contact.
🧠 Don't share, don't scrub, don't send back early.

πŸ“… What "getting better" looks like

  • 24–48 h: no new lesions appearing; existing sores start drying
  • 3–5 days: crusts loosening and falling away, redness fading
  • 7–10 days: skin healed, usually with no scar (temporary pink or pale patches are normal)
  • Not improving in 48 h? β€” reassess: wrong depth (ecthyma), resistant organism, poor adherence, or an untreated underlying condition such as scabies or eczema
🧠 "48 hours is the checkpoint." No improvement by then means something else is going on.
πŸ”€

TELL THEM APART

STEP 4 Β· THE REAL EXAM SKILL

Crusty and around the mouth is impetigo. Learn what it is NOT.

πŸ—ΊοΈ Where each one shows up

WHERE IT SHOWS UP β€” the real exam skill is telling these apart Same body, five patterns. Distribution narrows the answer before you ever read the description. CELLULITIS one leg, spreading, warm IMPETIGO around nose & mouth, hands TINEA scalp, trunk, groin, feet, nails SHINGLES ONE band, ONE side, stops at midline PSORIASIS EXTENSOR: elbows, knees, scalp, sacrum
🧠 Around the mouth + golden crust = impetigo. Same body, five patterns β€” distribution answers the question first.

πŸ“Š The look-alikes

CONDITIONHOW IT DIFFERS FROM IMPETIGO
Cold sore (HSV-1)A tight cluster of small vesicles on the lip border, preceded by tingling; recurs in the same spot. Impetigo is more scattered, oozier, and crusts golden.
CellulitisPainful, hot, swollen, spreading, often febrile, deep. Impetigo is itchy, superficial and crusty in a well child.
Eczema (atopic dermatitis)Chronic, dry, itchy, flexural. Watch out: eczema is a common portal of entry, so a child can have both β€” a suddenly weeping, crusted eczema patch is usually secondarily infected.
Tinea corporisRing with a scaly raised edge and a clear center. Fungal β€” treat with an antifungal, not an antibiotic.
Chickenpox / shinglesVesicles on a red base; chickenpox is widespread with fever, shingles is one dermatome. Viral β€” antivirals, not antibiotics.
ScabiesIntense night-time itch, burrows in web spaces, often several family members. Frequently gets secondarily infected with impetigo.
🧠 Golden crust = bacteria. Clustered blisters = virus. A ring = fungus. Night itch in the whole family = mites.

⭐ NCLEX traps

  • The crusting stage is the MOST contagious, not the least.
  • School exclusion is tied to treatment started + lesions drying, not to how the sores look alone.
  • Teaching must include separate laundry in hot water β€” a frequently-chosen wrong-if-omitted option.
  • The renal complication follows strep skin infection; rheumatic fever follows strep throat.
  • Ointment goes on clean skin after the crust is soaked off.
  • A painful, punched-out ulcer that scars is ecthyma β€” it needs an oral antibiotic.
🧠 If the option mentions hot water and separate laundry, look at it hard β€” it is usually correct.

🧠 One-line contrasts

  • Impetigo vs cellulitis β€” crusty & itchy vs hot & painful
  • Impetigo vs HSV β€” golden crust vs grouped vesicles that recur in one spot
  • Impetigo vs tinea β€” crust vs ring with a clear center
  • Non-bullous vs bullous β€” tiny vesicles vs big floppy blisters
  • Impetigo vs ecthyma β€” heals clean vs punches into the dermis and scars
🧠 Crust, cluster, ring, ulcer. Four words, four diagnoses.
⚑

QUICK RECALL

SAY IT OUT LOUD
🍯 Honey-colored crustChild · around nose & mouth · itchy.
🚨 Crusting = most contagiousContact + fomites: towels, linen, toys.
πŸ’Š Few = cream Β· Lots = pillMupirocin topically; oral for widespread, bullous or ecthyma.
🏫 5 S'sSchool out · Soap · Separate laundry · Short nails · Soak the crust.
🎯 Cover & check β€” 8 rapid-fire questions
Q1: Which organisms cause impetigo?
Staphylococcus aureus and Group A Streptococcus (Streptococcus pyogenes) β€” alone or together.
Q2: Describe the classic lesion and where it appears.
Itchy red sores that burst and dry into a stuck-on, honey-colored (golden) crust, classically around the nose and mouth of a young child; also hands and feet.
Q3: At which stage is impetigo most contagious?
When the vesicles rupture and crust β€” the oozing/crusting stage. A crusted lesion is still shedding bacteria.
Q4: List the five home/school teaching points.
1. Stay home from school/daycare until treated (about 24–48 hours) and lesions are drying. 2. Wash hands before and after touching the sores. 3. Wash the child's clothes, towels and linen separately in hot water. 4. Keep fingernails short and filed. 5. Gently soak and remove crusts before applying the ointment.
Q5: When would you expect an oral antibiotic rather than a topical one?
Widespread lesions, bullous impetigo, ecthyma, several household members affected, or failure of topical treatment. Topical mupirocin is used for a few localized lesions.
Q6: Which serious complication follows a streptococcal skin infection, and when?
Post-streptococcal glomerulonephritis, typically 1–3 weeks after the skin infection. Teach families to watch for tea/cola-colored urine, puffy eyes, reduced urine output, headache and high blood pressure.
Q7: A child has a painful, punched-out ulcer with a thick crust on the leg. What is it and what changes?
Ecthyma β€” impetigo that has extended into the dermis. It is painful, needs an oral antibiotic, and it will scar.
Q8: A parent asks whether they can just put the ointment straight over the crust. What do you say?
No β€” soften the crust first with a warm soapy soak and gently remove it, then apply the ointment to clean skin, or the medicine will not reach the bacteria. Wash hands before and after, and wear gloves if available.