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Nursing Field Notes / Labs & Values · Flagship priority page · Med-Surg + NCLEX

Labs — Highest Priority 🚨

Which abnormal result you act on FIRST — and exactly what you do about it

NG-194 LABS & VALUES · PRIORITY ADHD-friendly visual edition

This is the page to open before a clinical shift and before any “which client do you see first?” question. Every entry is built the same way: the value → what it does to the body → what you do about it. If you only ever learn four things from the whole labs batch, learn potassium, sodium, glucose and the ABG — those four sit behind most of the results that will make you pick up the phone.

📄 Simple Nursing original — opens in Drive →

🚨 ABC, then acuteAirway & breathing beat circulation. Circulation beats neuro. Acute beats chronic, unstable beats stable, actual beats risk.
💓 K⁺ 3.5–5.0Either direction is lethal. Peaked T = high. Flat T + U = low. First action either way: cardiac monitor.
🫁 60 and 50PaO₂ under 60 = hypoxemic failure. PaCO₂ over 50 = hypercapnic failure. A pulse oximeter cannot show you the CO₂.
🩸 S · A · P · RBleeding on an anticoagulant: Stop the drug · Assess for bleeding · Prep the antidote · Report.
⚠️ Read this before you memorize a single number. Every value on this page is a typical adult reference range — ranges vary by laboratory, so always use the range printed on your patient’s result. Critical-value cut-offs vary even more: every facility publishes its own critical list and its own call-back policy. Learn the direction and the meaning; verify the exact digits locally.
🚨

PART 1 · WHO DO YOU SEE FIRST

THE SORTING RULE

Before you compare numbers, sort the clients. The sort is always the same.

FIGURE 1 · THE A-B-C LADDER — drawn as the organs it protectsANTERIOR VIEW, cut away · read TOP to BOTTOM · each organ is tagged with the lab result that threatens it① airway → ② breathing③ circulation → ④ disabilityBRAINLUNGLUNGRALARVLVHEARTALVEOLUS ×400gas exchange happens hereTRACHEAmain bronchusleft ventricle → body1A + B · AIRWAY & BREATHINGthe lab that tells you: the ABGPaO₂ under 60 mmHg → hypoxemic respiratory failurePaCO₂ over 50 mmHg → hypercapnic respiratory failurepH under 7.35 with a rising CO₂ = they are tiring out2C · CIRCULATIONthe labs: K⁺ · Hgb · platelets · INR / aPTTK⁺ under 2.5 or over 6.5 mEq/L → lethal rhythmHgb under 7 g/dL → tissue cannot get its oxygenPlatelets under 20 ×10⁹/L · INR over 4 → bleeding3D · DISABILITY (the brain)the labs: glucose · sodiumGlucose under 50 mg/dL → the brain has no fuel NOWNa⁺ under 120 mEq/L → cerebral edema, seizuresNa⁺ over 160 mEq/L → shrivelled brain, agitationHOW TO USE THE LADDERWork DOWN it. An airway problem always outranks acirculation problem, and a circulation problem alwaysoutranks a neuro one — no matter how frightening theneuro number looks on the screen.WHAT THE PANEL COLORS MEANA/B — oxygen in, CO₂ outC — pump, carry, clotD — the brain’s fuel & water

Figure 1 — the A-B-C ladder drawn as the organs each letter protects, with the laboratory values that threaten each organ attached by leader lines. Work down it, never across it.

🚨 The one rule that answers most “who first?” questions

Airway and Breathing outrank everything. Then Circulation. Then Disability (neuro). A frightening neuro number never jumps ahead of a failing airway.

  • A/B — can they move air, and is the ABG telling you they cannot?
  • C — can the heart beat, the blood carry oxygen and the blood clot?
  • D — is the brain fueled (glucose) and correctly watered (sodium)?
🧠 A-B-C-D: Air · Blood · Clot · Consciousness. Sort every list of clients into those four buckets before you compare the numbers.

⭐ The four tie-breakers, in order

1ACUTE beats CHRONIC. A new potassium of 6.8 outranks a creatinine that has been 3.0 for two years.
2UNSTABLE beats STABLE. Trending in the wrong direction outranks a bad-but-steady number.
3ACTUAL beats RISK. A client who is bleeding outranks a client who might bleed.
4UNEXPECTED beats EXPECTED. A high glucose in a known diabetic is expected. A high glucose in a fasting post-op client is not.
🧠 “A-U-A-U” — Acute, Unstable, Actual, Unexpected. Say it out loud like a bird call. Four sorts, then pick.

📈 Read the TREND, not the snapshot

Answer first: a moving number beats a parked number.

A hemoglobin of 8.4 that was 11.2 this morning is far more urgent than a hemoglobin that has sat at 8.2 for a week. The same is true of creatinine, potassium and platelets.

Always ask: what was it last time, and how long ago?

🧠 A falling H&H is bleeding until proven otherwise. A rising creatinine is a kidney being injured right now.
FIGURE 2 · THE FOUR-CLIENT QUESTION — how to sort it in 20 secondsFOUR BEDS DRAWN LEFT → RIGHT · each bed carries its client’s data · the ranked answer is written under the bedsask: which one has anAIRWAY or CIRCULATION problem?ALow-grade fever, headacheand fatigue for 3 daysChronic · stable · no ABCthreatSEE 4thBSwollen, bruised left footafter a running accidentInjury · circulation intact ·stableSEE 3rdCAbdominal AND CHEST PAINafter a large spicy mealCHEST PAIN = rule out cardiacFIRSTSEE 1stDChild with a 10 cmlaceration to the chinBleeding — but controllable;not airwaySEE 2nd🎯 SEE CLIENT C FIRST — and here is the reasoning, in the order you should think it① Scan for AIRWAY and BREATHING first. Nobody here has an airway problem — so drop to CIRCULATION.② CHEST PAIN is a circulation problem until an ECG and troponin say otherwise. “Spicy meal” is a distractor: you cannot rule out an MI at the doorway.③ The chin laceration bleeds but is controllable with pressure — urgent, not first.④ The bruised foot is an injury with intact circulation — assess neurovascular status, but it waits.⑤ The three-day fever and headache is the most chronic and the most stable — it goes last.ACUTE beats chronic · UNSTABLE beats stable · ACTUAL problem beats risk-of-a-problem · and ABC beats everything.

Figure 2 — the classic four-client question drawn out. The reasoning panel underneath is the thinking you should be able to do in about twenty seconds.

🚨 Chest pain is the number-one priority symptom

Any chest pain — even with a perfectly good indigestion story — is treated as cardiac until an ECG and a troponin say otherwise.

Never let a plausible non-cardiac explanation talk you out of an ECG.

The same logic applies to: unrelieved pain under a cast (compartment syndrome), a systolic over 180 with symptoms (hypertensive crisis), and severe hypotension (shock).

🧠 “Lose life or limb.” Chest pain risks the life; compartment syndrome risks the limb. Both go to the front of the queue.

⚠️ Compartment syndrome — the limb emergency hiding in a pain score

The tell: pain that is out of proportion and NOT relieved by the ordered analgesia, in a limb with a cast, a splint, a fracture or a crush injury.

  • Pain on passive stretch of the fingers/toes
  • Paraesthesia (tingling) — an early sign
  • Pallor, pulselessness, paralysis — late signs; the limb is already in trouble

Do not elevate above heart level and do not apply ice — both reduce arterial flow. Call the provider.

🧠 The 6 P’s: Pain, Pressure, Paraesthesia, Pallor, Pulselessness, Paralysis. The first three are the ones that save the limb.

✅ What “see first” actually means at the bedside

Seeing a client first does not mean doing everything for them first. It means laying eyes on them and assessing.

  • Look, listen, and take a set of vital signs
  • Apply oxygen / a cardiac monitor if the picture calls for it
  • Then delegate, then document, then call

Assessment is almost always the correct first action — but not when the situation is already an emergency you can fix (a client who is choking, a stopped IV of insulin, an unconscious hypoglycemic client).

🧠 Assess first — unless there is an ABC you can fix right now. Then fix, then assess.
☠️

PART 2 · THE VALUES THAT KILL

VALUE → BODY → ACTION

Potassium, sodium and glucose. Three labs behind an enormous share of rapid responses.

FIGURE 3 · POTASSIUM ON THE MONITOR — the single most tested picture in labsFOUR RHYTHM STRIPS, same patient, four potassium states · read TOP → BOTTOM · lead II · calibrated ECG paperpeaked T = HIGH ⬆flat T + U = LOW ⬇1 mVpaper speed 25 mm/s · 1 large box = 0.20 s · 1 small box = 1 mmNORMAL · K⁺ 3.5–5.0 mEq/Lrounded upright T wave · narrow QRS · P before every QRSBASELINEnormal rounded Tnarrow QRS (under 0.12 s)1 mVpaper speed 25 mm/s · 1 large box = 0.20 s · 1 small box = 1 mmHYPERKALEMIA · K⁺ above 5.0 mEq/LTALL, NARROW, PEAKED (tented) T waves — the earliest change🚨 CALL THE PROVIDERTALL PEAKED “TENTED” TP wave flattening / disappearing1 mVpaper speed 25 mm/s · 1 large box = 0.20 s · 1 small box = 1 mmSEVERE HYPERKALEMIA · K⁺ climbing past ~6.5 mEq/LQRS WIDENS and blurs into the T — the next step is a sine wave, then arrest🚨 ARREST IS NEXTWIDE, SLURRED QRST merges with QRS → sine wave1 mVpaper speed 25 mm/s · 1 large box = 0.20 s · 1 small box = 1 mmHYPOKALEMIA · K⁺ below 3.5 mEq/LFLAT T wave · ST depression · a U wave appears after the T🚨 CHECK THE BREATHINGFLATTENED TU WAVE — “Uh-oh, K is low”ST depressionTRACE COLOR = POTASSIUM STATEnormal potassiumHIGH potassium (hyperkalemia)LOW potassium (hypokalemia)🧠 HIGH and PEAKED · LOW and FLAT with a UBoth directions are lethal. The direction changes the DRUG — it never changes the FIRST ACTION,which is: put the client on a cardiac monitor, then call the provider.

Figure 3 — four rhythm strips on calibrated ECG paper. If you can recognize the tented T and the flat-T-plus-U at a glance, you can answer most potassium questions without reading the stem twice.

🚨 HIGH potassium — the value, the body, the action

Value: above 5.0 mEq/L. Commonly critical at about 6.0–6.5 and above.

What it does: the resting membrane potential drifts up, so the heart first becomes irritable and then cannot repolarize. Tall peaked T waves → flat/absent P → widening QRS → sine wave → V-fib and arrest. Muscles tingle, then go weak; bowel sounds go hyperactive.

What you do: cardiac monitor first. Hold every potassium source — IV fluids with K⁺, oral supplements, salt substitutes, potassium-sparing diuretics, ACE inhibitors/ARBs. Recheck the level (a hemolysed specimen can fake a high K⁺). Then call.

🧠 “High and PEAKED.” Tall tented T = too much K. Think of a tent pitched on the strip.

💊 The hyperkalemia drug ladder — and why it is in that order

1IV calcium gluconate — does not lower the potassium. It protects the myocardium from the potassium while you work. Given first when the ECG is already changing.
2IV regular insulin with 50% dextrose — drives potassium into the cells. Fast, but temporary. Watch for hypoglycemia afterwards.
3Sodium polystyrene sulfonate (and newer binders) — removes potassium through the gut. Slow.
4Dialysis — removes it definitively. The answer when the kidneys are the problem, or when the level is extreme and the ECG is wide.

Ladder logic: protect → shift → remove. Every drug on this ladder is given per provider order — the nurse's independent first actions are the cardiac monitor and holding all potassium sources.

🧠 “C-I-B-D: Calcium, Insulin, Binder, Dialysis.” Protect the heart, shift it in, bind it out, dialyze it away.

🧪 LOW potassium — the value, the body, the action

Value: below 3.5 mEq/L. Commonly critical at about 2.5 and below.

What it does: everything gets low and slow. Flat T waves, ST depression, a U wave, PVCs. Weak legs, decreased reflexes, constipation and ileus — and the sign students miss, shallow respirations, because the diaphragm is a muscle.

What you do: assess the respirations and the rhythm. Check urine output before replacing. Replace on a pump.

🧠 “Flat T, and a U for Uh-oh.” The U wave is potassium waving goodbye.

❌ The potassium rule you can never break

Potassium is NEVER given IV push. Ever.

IV replacement is always diluted, always on an infusion pump, and never faster than the ordered rate — commonly a maximum of 10–20 mEq per hour through a peripheral line, and only with a rhythm monitored.

  • Burning at the site → slow the rate, do not stop the order; check the site and tell the provider.
  • Confirm urine output first — you cannot excrete what you cannot pee out.
  • Oral potassium goes with food to protect the stomach.
🧠 “K goes slow or the patient goes.” A potassium push is a cardiac arrest with a prescription attached.
FIGURE 4 · THE POTASSIUM TRAFFIC LIGHTone axis, low on the LEFT and high on the RIGHT · the consequence is written under the zone it belongs toCRITICAL LOWLOWNORMAL 3.5 – 5.0HIGHCRITICAL HIGH2.53.55.06.5mEq/LPOTASSIUM (K⁺) — “potassium PUMPS the heart”typical adult reference range · ranges vary by laboratory — use the range printed on your patient’s result⬅ LOW & SLOWflat T · ST depression · U waveSHALLOW BREATHING (the diaphragm is a muscle)weak legs · constipation · ileusHIGH & TIGHT, then LIMP ➡peaked T → wide QRS → V-fib · arresttingling then muscle weaknesshyperactive bowel · diarrheaNORMALnothing to report🚨 FIRST ACTION — either direction: cardiac monitor, then call the provider.NEVER IV-push potassium. Replacement is always diluted, always on a pump, and never faster than the ordered rate (commonly a maximum of 10–20 mEq per hour).

Figure 4 — one axis, low on the left, high on the right, with the clinical consequence written under the zone that causes it. Redraw this from memory and you own potassium.

🧂 CRITICAL sodium — the value, the body, the action

Value: normal 135–145 mEq/L. Danger usually below about 120 and above about 160.

What it does: water follows salt, so sodium is a brain lab. Low sodium → water moves into brain cells → cerebral edema → headache, confusion, seizures, coma. High sodium → water is pulled out → thirst, dry sticky tongue, flushed skin, agitation → seizures.

What you do: neuro checks and seizure precautions. Strict intake/output and daily weights. Fluid restriction for dilutional hyponatremia; hypertonic saline only per order, slowly, on a pump.

🧠 “Salt low = brain swells. Salt high = brain shrivels.” Either way you are watching level of consciousness.

🚨 The correction trap — the reason sodium is on this page twice

Correcting sodium too fast is its own emergency.

  • Raising a low sodium too quickly → osmotic demyelination (central pontine myelinolysis) — permanent neurological injury.
  • Lowering a high sodium too quickly → cerebral edema and seizures.

Never “catch up” a missed hypertonic saline rate, and never free-flow it.

Expect frequent serum sodium levels during correction — that is the monitoring, and it is your job to make sure they are drawn.

🧠 Sodium is corrected at the pace of a slow walk, not a run. The brain cannot re-equilibrate faster than that.
FIGURE 5 · SODIUM IS A BRAIN LAB — water follows saltTHREE CORONAL (front-to-back) SECTIONS · read LEFT → RIGHT · the skull is the SAME SIZE in all three — only the brain changeslow Na = brain SWELLShigh Na = brain SHRIVELS1LOW SODIUM · Na⁺ under 135water moves INTO brain cellsbrain stemventricles squeezed to slits · no CSF space left2NORMAL · Na⁺ 135–145water stays where it belongsSKULL — a fixed boxnormal slit-like ventricles3HIGH SODIUM · Na⁺ over 145water is pulled OUT of brain cellsCSF gap widens · bridging veins stretchWHAT EVERY COLOR IN THIS FIGURE MEANSbrain tissue (gray + white matter)lateral ventricle / CSF spaceskull — a fixed boxpressure pushing outwardHYPONATREMIA — “low and slow”headache → confusion → mental status changeseizures · coma · respiratory arrestnausea, cramps, fatigueWHAT YOU MONITOR EITHER WAYLOC and neuro checks — hourlyseizure precautions at the bedsidestrict intake/output + daily weightHYPERNATREMIA — “big, bloated, dry inside”extreme thirst · dry sticky tongueflushed skin · increased muscle tonerestless & agitated → seizures🚨 THE TRAP: correcting sodium TOO FAST is its own emergency.Raising a low sodium too fast risks osmotic demyelination; dropping a high sodium too fast risks cerebral edema. Slow · ordered · on a pump.

Figure 5 — the same skull three times. The skull is the fixed box; only the brain changes. That is the entire reason sodium is dangerous.

🍬 CRITICAL glucose — low kills faster than high

Value: typical adult fasting 70–110 mg/dL (many laboratories use the tighter fasting 70–99). Treat below 70; below 50–54 is a true emergency.

What it does: the brain stores no fuel. Shaky, sweaty, tachycardic and hungry first; then confused, slurred, combative; then seizure and coma.

What you do: conscious and able to swallow → 15 g of fast carbohydrate, recheck in 15 minutes. Unconscious or NPO → IV dextrose 50% or glucagon per order. Then a complex carbohydrate with protein so it does not fall again.

🧠 “15 and 15.” 15 grams, 15 minutes, recheck. Repeat up to the ordered limit, then escalate — do not keep stacking juice.

⭐ Hypoglycemia hides — three groups where it does not look like itself

  • Beta-blocker users — the shakiness and tachycardia are blunted. Sweating still happens, and that may be the only clue.
  • Older adults — it can present purely as confusion, a fall, or “not themselves.” Check a glucose on every acutely confused client.
  • Sleeping/night-shift clients — nightmares, sweating and a morning headache.

High glucose matters too, but it kills over hours (DKA/HHS), not minutes.

🧠 Any sudden change in behavior is a fingerstick until proven otherwise. It takes thirty seconds and it is free.
FIGURE 6 · GLUCOSE — the brain’s only fuelONE AXIS, low LEFT to high RIGHT · a neuron is drawn at each end showing what the number does to itDEADLYLOWNORMALHIGHCRITICAL5070110250mg/dLBLOOD GLUCOSEtypical adult fasting range 70–110 mg/dL (many labs use the tighter 70–99 fasting)⬅ HYPOGLYCEMIA — treat FIRSTshaky · sweaty · confused · slurredseizure → coma → brain injuryHYPERGLYCEMIA ➡thirsty · polyuria · blurred visionketones · Kussmaul breathing · DKANORMALbrain is fedSTARVED NEURONglucose under 50 mg/dLthe brain has NO stored fuelFED NEURONglucose 70–110 mg/dL🚨 WHICH ONE DO YOU TREAT FIRST? — the LOW one, every time.• A glucose of 40 mg/dL can kill in minutes. A glucose of 400 mg/dL kills over hours.• Conscious + able to swallow → 15 g of fast carbohydrate, recheck in 15 minutes (the “15/15 rule”).• Unconscious or NPO → IV dextrose 50% or glucagon per order, then recheck.• Then feed a complex carbohydrate + protein so it does not drop straight back down.• Rebound rule: never treat a low with insulin, and never stack treatments without rechecking.

Figure 6 — glucose on one axis with a starved neuron at one end and a fed neuron at the other. The center panel is the treatment sequence.

🫁

PART 3 · THE ABG THAT MEANS FAILURE

60 AND 50

Two numbers define respiratory failure. Learn them as a pair and you will never mix them up.

FIGURE 7 · THE TWO KINDS OF RESPIRATORY FAILURE — an ABG tells you whichBOTH PANELS SHOW THE SAME ALVEOLUS AND ITS CAPILLARY, magnified ×400 · read TOP → BOTTOMa pulse oximeter cannot show youthe CO₂ — only an ABG can1HYPOXEMIC FAILURE — “not enough oxygen IN”the number that defines it: PaO₂ under 60 mmHg · the SpO₂ will fall tooterminal bronchioleALVEOLUSair sacblood flowPULMONARY CAPILLARYmembrane THICKENED by fluidalveolar–capillary membraneO₂ INCO₂ OUTWHY IT HAPPENSFluid, pus or blood filling the sac — pneumonia,pulmonary edema, ARDS, drowningA clot cutting off the capillary — PEA collapsed sac — atelectasis, pneumothoraxWHAT YOU SEERestless → agitated → confused (earliest signs)Pale, dusky, cool and clammy skinTachypnoea, tachycardia, accessory musclesCyanosis is a LATE sign — never wait for it2HYPERCAPNIC FAILURE — “not enough CO₂ OUT”the number that defines it: PaCO₂ over 50 mmHg · the SpO₂ can look fineterminal bronchioleALVEOLUSair sacblood flowPULMONARY CAPILLARYalveolar–capillary membraneO₂ INCO₂ OUTWHY IT HAPPENSThe bellows are failing — COPD, severe asthmaThe drive is suppressed — opioids, sedativesThe muscles cannot pull — Guillain-Barré,myasthenic crisis, high spinal cord injuryWHAT YOU SEEDrowsy → hard to rouse (CO₂ narcosis)Morning headache, flushed warm skinShallow, slowing respirationsA “quiet” tiring patient is the dangerous oneWHAT EVERY ARROW AND COLOR MEANSoxygen crossing normallycarbon dioxide leaving normallygas BLOCKED — ✕ means it cannot crosspulmonary capillary + red blood cellsLOWNORMAL 35–45HIGHFAILURE354550mmHgPaCO₂ — how much carbon dioxide is left behind⬅ blowing it off(anxiety, pain, early sepsis)over 50 = hypercapnic failure ➡drowsy · headache · flushedFAILURELOWNORMAL 80–100HIGH6080100mmHgPaO₂ — how much oxygen is dissolved in the blood⬅ under 60 = hypoxemic failurerestless · pale, dusky · cool & clammyusually already on O₂ ➡🚨 WHAT YOU ACTUALLY DO — in order1Sit them UP, apply oxygen, and stay in the room. Position and oxygen come before charting.2Look at the client, not just the number: restless, agitated, pale, dusky, cool and clammy = hypoxia.3Rising CO₂ with a falling pH → non-invasive ventilation (BiPAP) is the usual next step per order.4If they cannot protect the airway, or BiPAP fails → intubation and mechanical ventilation.

Figure 7 — one alveolus and its capillary, drawn twice. Oxygen failing to get in is a different emergency from carbon dioxide failing to get out, and only an ABG separates them.

🧪 The four ABG numbers you must know cold

pH7.35 – 7.45acid ⟵ ⟶ alkaline
PaCO₂35 – 45 mmHgthe RESPIRATORY number
HCO₃⁻22 – 26 mEq/Lthe METABOLIC number
PaO₂80 – 100 mmHgoxygenation

Typical adult reference ranges — verify against the ranges printed on your own result.

🧠 CO₂ is the lungs. HCO₃ is the kidneys. Whichever one moves the same way as the pH is compensating; the other one caused the problem.

📈 ROME — the two seconds that sort any ABG

Respiratory Opposite · Metabolic Equal.

pHCO₂ / HCO₃Answer
⬇ lowCO₂ ⬆ highRespiratory acidosis
⬆ highCO₂ ⬇ lowRespiratory alkalosis
⬇ lowHCO₃ ⬇ lowMetabolic acidosis
⬆ highHCO₃ ⬆ highMetabolic alkalosis

Respiratory problems move the pH and the CO₂ in opposite directions; metabolic problems move the pH and the bicarbonate in the same direction.

🧠 ROME. Respiratory Opposite, Metabolic Equal. Write it on the corner of your scratch paper before the exam starts.

🚨 PaO₂ under 60 — hypoxemic respiratory failure

What it does: tissues stop getting oxygen. The brain shows it first: restless → agitated → confused. The skin goes pale, dusky, cool and clammy. Cyanosis is a late sign.

What you do: sit them up, apply oxygen, stay in the room, and call. Expect a higher-flow device or non-invasive ventilation per order.

Never label a newly restless, agitated client as “anxious” until you have ruled out hypoxia.

🧠 Restlessness is the earliest sign of hypoxia. The agitated client is not being difficult — they are drowning.

🚨 PaCO₂ over 50 — hypercapnic respiratory failure

What it does: carbon dioxide is an acid. It builds up, the pH falls, and the client becomes drowsy and hard to rouse (CO₂ narcosis) with a headache and warm flushed skin.

What you do: the quiet, sleepy, tiring client is the dangerous one. Escalate: BiPAP is the usual next step per order; if they cannot protect the airway or BiPAP fails, intubation and mechanical ventilation.

🧠 A tiring patient goes quiet before they stop. Falling respiratory rate in someone who was working hard is not improvement.

⚠️ The pulse-oximeter trap

SpO₂ measures oxygen only. It says nothing about carbon dioxide.

A client on oxygen can hold a comfortable-looking SpO₂ 94% while their PaCO₂ climbs to 70 and their pH falls. That is why the question stem gives you an ABG and not a probe reading.

The probe also lies with: poor perfusion, cold hands, motion, nail polish, and carbon monoxide poisoning (falsely normal or high).

🧠 “Sat 94%” is not the same as “ventilating.” Oxygenation and ventilation are two different jobs and two different numbers.
🩸

PART 4 · COUNTS, CLOTS AND BLEEDS

WHITE · PLATELET · COAG

The counts that mean the client cannot fight infection, and the numbers that mean they cannot stop bleeding.

FIGURE 8 · NEUTROPENIA — the low number that makes a low-grade fever an emergencyMAGNIFIED BLOOD FIELD ×1000 · LEFT = enough neutrophils · RIGHT = too few · the bacteria are drawn to the same scale in both fieldsNORMAL DEFENSE · WBC ~4.5–11.0 ×10⁹/Lplenty of neutrophils — bacteria get eatenNEUTROPENIA · ANC under 500 cells/µLalmost no neutrophils — bacteria multiply unopposed🚨 a fever here is a MEDICAL EMERGENCYWHAT EACH CELL ISneutrophil — the bacteria-eaterbacteriared blood cells (background)THE ANCabsolute neutrophil countANC = WBC ×(% neutrophils + % bands)÷ 100WORKED EXAMPLEWBC 2,000 /µLsegs 18% + bands 4% = 22%ANC = 2,000 × 0.22 = 440→ SEVERE NEUTROPENIAover 1500normal defense1000–1500mild500–1000moderateunder 500SEVERE · isolate🚨 NEUTROPENIC PRECAUTIONS — and why a low-grade fever counts• A neutropenic client cannot make pus, cannot make redness and cannot mount a big fever — the usual signs of infection are missing.• So a temperature of 100.4 °F (38 °C) is treated as SEPSIS until proven otherwise: cultures, call the provider, antibiotics fast.• Protective precautions: private room · strict hand hygiene · no fresh flowers or standing water · no raw fruit or vegetables · no sick visitors.• No rectal temperatures, no suppositories, no enemas — the rectal mucosa tears and lets bowel bacteria straight into the blood.

Figure 8 — the same magnified blood field with plenty of neutrophils and with almost none. The bacteria are drawn to the same scale in both, so the difference is the defense, not the infection.

🚨 Severe neutropenia — a fever here is the emergency

Value: total WBC below about 5,000/mm³ is leukopenia; the number that actually matters is the ANC below 500 cells/µL.

What it does: without neutrophils the client cannot make pus, cannot make redness and cannot mount much of a fever — the usual signs of infection are missing. Infection goes from unnoticed to septic very quickly.

What you do: a temperature of 100.4 °F (38 °C) is treated as sepsis until proven otherwise — cultures, call the provider, antibiotics fast. Protective precautions stay up.

🧠 “Low grade fever = KILL.” In a neutropenic client a small fever is the whole warning you are going to get.

✅ Neutropenic precautions — the list, and the two nobody remembers

  • Private room; strict hand hygiene by everyone entering
  • No fresh flowers, no potted plants, no standing water
  • No raw fruit or vegetables, no undercooked food (a “low-bacteria” diet per facility policy)
  • No sick visitors, no crowds; mask the client if they must leave the room
  • No rectal temperatures, suppositories or enemas — the mucosa tears and lets bowel bacteria into the blood
  • Meticulous oral care with a soft brush; inspect IV sites every shift

Causes to expect on the chart: chemotherapy, immunosuppressants, steroids, HIV, autoimmune disease.

🧠 Nothing raw, nothing rectal, nothing crowded. Three “no”s cover most of the exam questions on this topic.
FIGURE 9 · THE BLEEDING NUMBERS — platelets, INR and aPTTTOP: the SAME torn vessel drawn twice, cut-away side view · LEFT = the plug holds · RIGHT = the plug failsplug = PLATELETSmesh = CLOTTING FACTORS1ENOUGH PLATELETS + NORMAL CLOTTINGplatelets plug the hole, fibrin mesh seals itblood flowTEAR in the vessel wall✅ PLUG HOLDS — the bleeding stopsplatelet plug + fibrin mesh2TOO FEW PLATELETS / TOO MUCH DRUGnothing to plug with, nothing to seal withblood flowTEAR in the vessel wall🚨 PLUG FAILS — the bleeding continuesblood escapes the vesselWHAT EVERY MARK IN THE TOP PANELS MEANSplatelet — “platelets PLUG the bleeding”fibrin mesh — built by the clotting factorsred blood cellblood escaping through the tearmuscular vessel wall (media)outer fibrous coat (adventitia)<20<50LOWNORMAL 150–4002050150PLATELETS×10⁹/L (150,000–400,000/mm³)⬅ under 20: spontaneous bleedingunder 50: bleeds with minor traumaUNDER 2.0THERAPEUTICHIGHDANGER2.03.04.0INR — watches WARFARINno units · untreated 0.9–1.2 · usual target on warfarin 2.0–3.0over 4: hold the dose ➡antidote = vitamin K30–40 sTHERAPEUTICHIGHDANGER4070100aPTT — watches HEPARINseconds · therapeutic ≈1.5–2.5× controlover 100 s: stop the drip ➡antidote = protamine sulfate🚨 THE FOUR-STEP BLEEDING RESPONSE — same four steps every time: S · A · P · R1. STOPhold the drug2. ASSESSlook for bleeding3. PREPget the antidote ready4. REPORTcall the provider

Figure 9 — the same torn vessel with a working plug and a failing one, then the three numbers that predict which of the two you are about to get.

🚨 Critical platelet counts — what each threshold means

150–400×10⁹/L
Normal. Also written 150,000–400,000/mm³.
under 150LOW
Watch for bruising, petechiae, bleeding gums. Review every drug that thins the blood.
under 50BLEEDS EASILY
Bleeding with minor trauma. Bleeding precautions on. Procedures usually held.
under 20SPONTANEOUS
Bleeding with no trauma at all, including intracranial. Expect a transfusion order.
🧠 “Platelets PLUG the bleeding.” Below 50 the plug is thin; below 20 there is no plug.

💊 INR over 4 and aPTT over 100 — the two anticoagulant alarms

INR — watches WARFARINUntreated 0.9–1.2 · usual target on warfarin 2.0–3.0 · over 4 = danger. Antidote: vitamin K (phytonadione).
aPTT — watches HEPARINBaseline about 30–40 s · therapeutic roughly 1.5–2.5× the control (often quoted 46–70 s) · over 100 s = danger. Antidote: protamine sulfate.

Low-molecular-weight heparin (enoxaparin) is not monitored with routine aPTT — an anti-Xa level is used when monitoring is needed.

🧠 “WarK-IN and He-PTT.” Warfarin has the IN of INR inside it; heParin has the PTT inside it.

✅ Bleeding on an anticoagulant — S · A · P · R

1STOP or hold the drug. Stop the infusion; do not give the next dose.
2ASSESS for bleeding — gums, nose, IV sites, urine, stool, vomit, bruising, abdominal or flank pain, a change in level of consciousness.
3PREP the antidote so it is ready when the order arrives.
4REPORT to the provider with the value, the trend and what you are seeing.

Bleeding precautions: soft toothbrush, electric razor, no flossing, avoid IM injections, hold pressure longer after any needle stick, fall precautions, no NSAIDs or aspirin unless ordered.

🧠 S-A-P-R — Stop, Assess, Prep, Report. Same four steps for warfarin, heparin and any other bleeding emergency.

🩸 Hemoglobin under 7 — the transfusion line

Value: typical adult roughly 12–18 g/dL overall — about 12–16 for women and 14–18 for men.

  • 8–11 — risky. Report it, especially before surgery.
  • under 7 — expect a transfusion order in most adult situations.

What it does: less carrier means less delivered oxygen — pale skin, cool clammy skin, fatigue and weakness, tachycardia and shortness of breath on effort.

🧠 “Below 7 → heaven or a transfusion.” Blunt, but you will never forget the number.
GO DEEPERFull red-cell detail on NG-121 · CBC.

🧪 Creatinine over 1.3 — the kidney is already hurt

Value: typical adult 0.6–1.2 mg/dL. Over 1.3 means a damaged kidney; a creatinine that is rising day on day is an acute injury happening now.

Pair it with urine output: less than 30 mL/hr is kidney distress and needs reporting.

Why it belongs on a priority page: a failing kidney is the most common reason a client ends up hyperkalemic — and it changes the dose of a great many drugs.

🧠 Creatinine is the kidney’s own handwriting. BUN can be pushed around by dehydration and protein; creatinine mostly cannot.
GO DEEPERFull renal detail on NG-048 · Renal labs.
📞

PART 5 · MAKE THE CALL

REPORT IT PROPERLY

You found it. Now say it in a way that gets an order in under two minutes.

📞 Critical value call-back — the part that is actually policy

1The laboratory phones a critical value to a licensed person. Write it down.
2Read it back and get confirmation. This is a National Patient Safety Goal, not a courtesy.
3Go and look at the client — does the number fit what you see?
4Notify the provider, document the notification and the time, and carry out the orders.

If the number does not fit the client at all, say so — but never delay the call while you wait for a redraw.

🧠 Write it, read it back, look at the patient, call. In that order, every time.

🗣️ SBAR for a critical lab — a script you can borrow

  • Situation — “Mr Alvarez in 412, his potassium came back at 6.8.”
  • Background — “He is a CKD stage 4 patient, missed dialysis yesterday, on lisinopril.”
  • Assessment — “He is on the monitor now; I see peaked T waves and his rate is 54. He is lethargic.”
  • Recommendation — “I have held his ACE inhibitor and all potassium. Do you want calcium gluconate and insulin with dextrose, and shall I call dialysis?”
🧠 Have the recommendation ready. A nurse who calls with a plan gets orders faster than a nurse who calls with a number.

⚠️ Before you call — three things that fake an abnormal result

  • Hemolysis — a traumatic draw bursts red cells and falsely raises potassium. Classic on a difficult stick or a squeezed fingerstick.
  • Drawing above an IV — dilutes the sample and drags the result toward whatever is in the bag. Draw from the opposite arm.
  • Wrong tube or a clotted tube — a clotted lavender tube gives a false low platelet count.

Suspecting a bad specimen does not mean you sit on the result. Report it and redraw per policy at the same time.

🧠 Question the specimen, never the phone call. Redraw and report — both.
GO DEEPERTube colors and draw order on NG-042 · BMP & electrolytes.

⭐ Which of these do I report? — the quick filter

ResultReport?
Hemoglobin 6 g/dL✅ Yes — transfusion territory
Potassium 6.5 mEq/L✅ Yes — rhythm emergency
Sodium 150 mEq/L✅ Yes — neuro risk
WBC 2,000/mm³✅ Yes — cannot fight infection
Platelets 45,000/mm³✅ Yes — bleeds with minor trauma
Temp 100.4 °F in a neutropenic client✅ Yes — treat as sepsis
BUN 19 mg/dL❌ No — still within 10–20
🧠 Do not get baited by a number just because it sits near the edge. In range is in range.

🧠 How to memorize this page in one sitting

  • Draw the potassium strip three times: normal, peaked, flat-with-U.
  • Say the two ABG thresholds out loud: “sixty and fifty.”
  • Say the two antidotes as a pair: “warfarin–vitamin K, heparin–protamine.”
  • Say the four sorts: “acute, unstable, actual, unexpected.”
  • Then cover the page and answer the eight questions below.
🧠 Five things, five minutes, out loud. Recall beats re-reading every single time.

🎯 Cover & check — 8 rapid-fire questions

Q1. Four clients arrive together. Who do you see first: the 3-day low-grade fever, the bruised foot, the chest pain after a spicy meal, or the chin laceration?
Show answerThe chest pain. Any chest pain is cardiac until an ECG and troponin say otherwise. Circulation outranks the rest.
Q2. The monitor shows tall, narrow, peaked T waves. Which electrolyte, which direction, and what is your first action?
Show answerPotassium, HIGH. First action: cardiac monitor and hold all potassium sources, then call. Anticipate calcium gluconate → insulin with dextrose → a binder or dialysis.
Q3. PaO₂ 55, PaCO₂ 42. Which kind of respiratory failure?
Show answerHypoxemic — the oxygen is failing (under 60) while the CO₂ is still normal. Sit them up, oxygen, call.
Q4. A client on warfarin has an INR of 4.5. What are your four steps?
Show answerStop/hold the warfarin · assess for bleeding · prep vitamin K · report to the provider.
Q5. A client on a heparin drip has an aPTT of 110 seconds. Which antidote do you prepare?
Show answerProtamine sulfate. Stop the drip first.
Q6. WBC 2,000/mm³ with 18% segs and 4% bands. Calculate the ANC and say what it means.
Show answerANC = 2,000 × (18 + 4) ÷ 100 = 440severe neutropenia. Protective precautions; a fever is a medical emergency.
Q7. Glucose 42 in a client who is drowsy and cannot safely swallow. What do you do?
Show answerIV dextrose 50% (or glucagon if there is no IV access), per order — never oral fluids in someone who cannot protect their airway. Recheck in 15 minutes.
Q8. Sodium 118. What two safety measures go up before anything else happens?
Show answerSeizure precautions and frequent neuro checks / LOC monitoring. Then fluid restriction or hypertonic saline per order — slowly, on a pump.
🚨 K⁺ either wayPeaked T = HIGH. Flat T + U = LOW. Cardiac monitor first, both times. Never IV push potassium.
🧂 Na⁺ is a brain labLow = brain swells, seizes. High = shrivels, agitated. Correct it SLOWLY — fast correction is its own emergency.
🫁 60 / 50PaO₂ under 60 = hypoxemic failure. PaCO₂ over 50 = hypercapnic failure. Restlessness is the earliest hypoxia sign.
🩸 Bleeding & countsINR > 4 · aPTT > 100 · PLT < 20 · ANC < 500 · Hgb < 7 · glucose < 50. Every one of these is a phone call.