What this page covers, and what it doesn't.NG-029 is the pathophysiology.
NG-109 is the complications and the labs.
This page is the care page: paracentesis and liver biopsy step by step, the drug map, the diet and fluid
rules, bleeding and itching precautions, and exactly what you teach before discharge. Nothing here cures cirrhosis
β every intervention on this page treats a complication or prevents the next one.
π§ βbIopsy β RIGHT side.β The liver is on the right, so you lie on the right and let
your own body weight hold the pressure.
β Paracentesis β the FIRST action, and why
βWhich action should the nurse take first?β
Have the patient empty their bladder. The needle goes into the lower abdomen, and a full bladder sits
right in that path. Puncturing it is exactly the complication the step prevents.
Then, in order: baseline vital signs β weight and abdominal girth (mark the spot) β position upright β
consent and sterile set-up.
π§ Bladder before needle. Every time, no exceptions.
π¨ After a paracentesis β what you are watching for
Hypotension and tachycardia β taking liters off fast shifts volume out of the circulation.
Leakage at the puncture site β apply a dry sterile dressing and monitor it.
Signs of infection or bowel injury β fever, worsening abdominal pain, rigid abdomen.
Re-measure weight and girth and compare with the pre-procedure figures.
Record the fluid β amount, color and clarity. Cloudy fluid suggests infection.
IV albumin is often ordered after a large-volume tap to keep the circulation from collapsing.
π§ Take fluid off β pressure can drop. Vital signs before AND after, always.
π§ͺ The other tests you will see ordered
TEST
WHAT IT ANSWERS
Liver function panel
ALT, AST, bilirubin, albumin β damage and function
Coagulation panel
PT, PTT, INR, platelets β can this patient clot?
Serum ammonia
Supports hepatic encephalopathy; trend it
Abdominal ultrasound
Liver texture and size, ascites, portal flow, spleen size
Upper endoscopy
Looks directly at varices β and can band them
Ascitic fluid analysis
Cell count and culture β is this SBP?
π§ Coagulation studies come BEFORE any invasive procedure in a liver patient.
β οΈ Before ANY invasive procedure here
Check PT, PTT, INR and platelets β and report abnormal results before the procedure starts.
Confirm consent and a valid time-out.
Establish IV access and know where the blood products are.
Take baseline vital signs β you cannot detect a change without them.
Anticipate that bleeding will be the complication, not infection.
π§ Every hole you make in this patient is a hole that keeps bleeding.
π
THE DRUG MAP
STEP 2 Β· PHARMACOLOGY
Match the drug to the complication it treats, and the monitoring writes itself.
πΊοΈ Which drug for which problem
π§ Never memorize a cirrhosis drug on its own β memorize it attached to the
complication it exists to fix.
π§ βLose the ammonia Β· Loose bowels Β· Lose potassium.β Three L's, one drug.
π °οΈ IV albumin β the one students get wrong
π§ Albumin RAISES the blood pressure. Bounding pulses and a rising BP mean it is working
β not that something has gone wrong. Keep listening to the lungs anyway.
π§ Diuretics in cirrhosis
An aldosterone antagonist (spironolactone) is the usual first choice, because aldosterone excess is
one of the three drivers of ascites. A loop diuretic is often added.
Aldosterone antagonistPotassium-SPARING β watch for HYPERkalemia.
Loop diureticPotassium-WASTING β watch for HYPOkalemia.
Together they can partly balance each other, but the potassium still has to be
checked. Potassium reference 3.5β5.0 mEq/L.
Monitor daily weight, girth, I&O, blood pressure and electrolytes.
Diuresing too fast can trigger encephalopathy through dehydration and electrolyte shifts.
The liver needs vitamin K to activate several clotting factors, so it may be given to try to improve
the PT/INR. It does not work if there are no liver cells left to use it.
Fresh frozen plasma replaces clotting factors when bleeding or before a procedure.
Platelets are given for severe thrombocytopenia with bleeding.
Packed red cells for significant blood loss.
All blood products follow your facility's transfusion policy, with the required checks and monitoring.
π§ Vitamin K is the ingredient; the liver is the kitchen. No kitchen, no clotting factors.
π½οΈ
DIET & FLUID
STEP 3 Β· NUTRITION
Low sodium, controlled fluid, adequate calories, and absolutely no alcohol.
π½οΈ The cirrhosis plate
π§ βLow sodium, Low swelling.β Salt holds water; water becomes ascites.
π§ Sodium β the practical teaching
No salt shaker at the table and none added in cooking.
Read labels β the sodium is mostly hidden in processed food, not in the shaker.
Use herbs, lemon, garlic and pepper for flavor instead.
Check with the provider before using a salt substitute β many are potassium chloride, which
matters if the patient is on a potassium-sparing diuretic.
π§ βThe salt is in the box, not the shaker.β Labels beat willpower.
π₯ Protein β say βask the providerβ, not βrestrict itβ
Protein is broken down into ammonia, so older teaching restricted it in everyone with cirrhosis. Current
practice is more nuanced:
These patients are usually malnourished and losing muscle, and muscle actually helps clear ammonia.
So protein is generally kept adequate, and restricted only in specific situations.
In active hepatic encephalopathy, a dietary consult to limit protein is a classic exam answer
β and remains part of the picture on many exams.
Safest position for the nurse: follow the ordered diet and get a dietitian involved.
π§ If the question says βencephalopathy β limit which ingredient?β, the answer is PROTEIN.
In real practice, ask the dietitian.
π§ Fluid restriction β how to make it bearable
Spread the allowance across the day and save some for the evening.
Use small cups β the same volume feels like more.
Ice chips count as roughly half their volume; frozen fruit pieces last longer.
Frequent mouth care and sugar-free hard candy or gum for dry mouth.
Avoid salty food β it makes the thirst much worse.
Include everything liquid: soup, jelly, ice cream, IV medications.
π§ Thirst is driven by SALT. Cutting sodium is what makes a fluid restriction tolerable.
π½οΈ Preventing malnutrition
Oral care BEFORE meals β this is the classic exam answer for improving intake.
Small, frequent meals β a full ascitic abdomen makes large meals impossible.
A bedtime snack helps, because a cirrhotic liver has poor glycogen stores overnight.
Supplements and vitamins as ordered β thiamine and folate are commonly needed, especially with
alcohol-related disease.
Weigh regularly, but remember that ascites hides real weight loss.
π§ Big belly, wasting muscles. The scale can look stable while the patient is starving.
π« Alcohol β the absolute rule
Complete abstinence, permanently, whatever the original cause of the cirrhosis was. There is no safe amount
for a liver that is already scarred.
Offer referral to a support program rather than a lecture.
Watch for alcohol withdrawal in a newly admitted patient β it can start within hours and is
dangerous in someone who is already confused.
Check mouthwashes, cough syrups and tonics for alcohol content.
π§ Scar tissue never comes back. Stopping alcohol protects the liver cells that are left.
ALT/AST for liver cell damage and lactate for tissue perfusion β including the detail that lactate is drawn without a tourniquet and put on ice. — swipe it sideways if it is cut off, or tap to open it full size.
Saved study graphics from your own collection. Each one is someone else’s work — check anything clinical against your course materials before you rely on it.